This pattern of symptoms suggests the possibility of:
BPPV Ménière’s Disease Lateral Medullary infarct
Vestibular neuritis
The sudden onset of severe vertigo and spontaneous nystagmus (observed by the MD in the Emergency Department) in this patient was likely the result of:
Otoconia in the semi-circular canal An alteration in the tonic firing rate of the vestibular nuclei
Otoconia attached to the cupula An alteration in the firing rate of the hair cells due to a structural abnormality
Which 2 performance-based measures at the body function and structure level of the ICF are valid for use with individuals with vestibular disorders?
Dizziness Handicap Inventory and Dynamic Gait Index Gait velocity and Dynamic Gait Index CTSIBm and Sharpened Rhomberg CTSIBm and Five Times Sit-to-Stand test
Recovery of gaze stability in individuals with unilateral incomplete loss of vestibular function is believed to occur due to:
Increasing the gain of the vestibulo-ocular reflex and re-growth of damaged hair cells Increasing the gain of the vestibulo-ocular reflex and substitution of alternative strategies
Substitution of alternative strategies Habituation and re-innervation of the vestibular nerve
Resolution of spontaneous nystagmus within 24 hours after unilateral vestibular damage occurs as a result of:
Spontaneous recovery at the level of the vestibular nuclei
Cessation of head movement Performing gaze stability exercises Otoconia being removed from the semi-circular canal
Case Scenario: You are treating a patient in a rehabilitation