Cellular level
Ty’Leasha Mendoza
Unit 2. Case Study 1 The Cellular Level of Organization
September 29, 2013 Joseph does have a few risk factors; stress on his body from long hours of work, he was a previous smoker, tightness in his chest, and increase in his weight and also has a family history of vascular disease. Joseph and his son were playing catch when his dad suddenly fails unconsciously to the ground and stopped breathing, his face changed to an ashy gray color and Marcus Joseph son called for help.
Joseph had several risk factors affecting his cellular function. The cells in his heart struggled to work, the blood flow slowed down along with oxygen, glucose and ions that were required for the heart to pump, the cells received more carbon dioxide causing his pH level to drop. The cells had to work twice as hard and fast, using more energy and oxygen than normal, eventually beginning to cause the cells to die.
Both intracellular organelles have membranes. The lack of ATP affects the plasma membrane and that special calcium ATPases had stopped moving calcium from cytosol into the endoplasmic reticulum of his cardiac muscle cells The breakdown of the membranes of these structures affect the functions of his heart cells because they keep everything in order, helps to keep organelles organized and enables the movement of the cell organelles. The instructions Josephs body needs to repair itself and his predispositions for the vascular diseases are both contained within the cell on the “power house of the cell” the Mitochondria structure.
Joseph heart attack caused the function of his cells to change. I believe the ribosomes, the endoplasmic reticulum, Golgi complex and lysosomes were the proteins in the cell membrane that were involved in the homeostatic imbalances of Joseph’s heart.
Reestablishing oxygen flow to Joseph’s body was so important because without oxygen to the cells they can not produce ATP, the energy of the cell. He was lucky enough to have
Unit 2. Case Study 1 The Cellular Level of Organization
September 29, 2013 Joseph does have a few risk factors; stress on his body from long hours of work, he was a previous smoker, tightness in his chest, and increase in his weight and also has a family history of vascular disease. Joseph and his son were playing catch when his dad suddenly fails unconsciously to the ground and stopped breathing, his face changed to an ashy gray color and Marcus Joseph son called for help.
Joseph had several risk factors affecting his cellular function. The cells in his heart struggled to work, the blood flow slowed down along with oxygen, glucose and ions that were required for the heart to pump, the cells received more carbon dioxide causing his pH level to drop. The cells had to work twice as hard and fast, using more energy and oxygen than normal, eventually beginning to cause the cells to die.
Both intracellular organelles have membranes. The lack of ATP affects the plasma membrane and that special calcium ATPases had stopped moving calcium from cytosol into the endoplasmic reticulum of his cardiac muscle cells The breakdown of the membranes of these structures affect the functions of his heart cells because they keep everything in order, helps to keep organelles organized and enables the movement of the cell organelles. The instructions Josephs body needs to repair itself and his predispositions for the vascular diseases are both contained within the cell on the “power house of the cell” the Mitochondria structure.
Joseph heart attack caused the function of his cells to change. I believe the ribosomes, the endoplasmic reticulum, Golgi complex and lysosomes were the proteins in the cell membrane that were involved in the homeostatic imbalances of Joseph’s heart.
Reestablishing oxygen flow to Joseph’s body was so important because without oxygen to the cells they can not produce ATP, the energy of the cell. He was lucky enough to have