Ribonucleocapsid are then released in the cytoplasm once the fusion of the membrane has occurred (Bressem 3). Both the H glycoprotein and the F glycoprotein can be used to interact with cellular receptors that are used to allow the entry of the virus, along with determining the susceptibility of the host, tissue tropism, and viral pathogenesis (Bressem 3). Replication of the virus occurs after there are enough nucleoproteins present in the cell to encapsulate “neosynthetized antigenomes and genomes” (Maruyama 2-3). The replication type that occurs within a cetacean morbillivirus and its host cell follows the negative-stranded RNA virus replication model. A virion (virus particle) is then released through the intereaction between the ribonucleocapsid and the matrix protein under the plasma membrane (Bressem 3). The replicated virus is then released from the cell using the budding method (Maruyama 2). These cycle continues as the spread of the virus increases and continues. Once the cetacean morbillivirus leaves the host cell, it does not survive very long in natural environments, meaning that the life of the virus after infection has already occurred is
Ribonucleocapsid are then released in the cytoplasm once the fusion of the membrane has occurred (Bressem 3). Both the H glycoprotein and the F glycoprotein can be used to interact with cellular receptors that are used to allow the entry of the virus, along with determining the susceptibility of the host, tissue tropism, and viral pathogenesis (Bressem 3). Replication of the virus occurs after there are enough nucleoproteins present in the cell to encapsulate “neosynthetized antigenomes and genomes” (Maruyama 2-3). The replication type that occurs within a cetacean morbillivirus and its host cell follows the negative-stranded RNA virus replication model. A virion (virus particle) is then released through the intereaction between the ribonucleocapsid and the matrix protein under the plasma membrane (Bressem 3). The replicated virus is then released from the cell using the budding method (Maruyama 2). These cycle continues as the spread of the virus increases and continues. Once the cetacean morbillivirus leaves the host cell, it does not survive very long in natural environments, meaning that the life of the virus after infection has already occurred is