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How Neurological Processes Affect Behaviour and Impact the Field of Biological Psychology

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How Neurological Processes Affect Behaviour and Impact the Field of Biological Psychology
How neurological processes affect behaviour and impact the field of biological psychology

Psychology as a field of study has evolved from the human need to understand ourselves and the causes behind our behaviours. Philosophy tried its hand first, attempting to describe it as a complex interaction between the mind and the body – between the mental and the physical. This basic idea resonates through this field of exploration through the years. While there have been diversions from this line of thought, tending towards behavioural or psychoanalytic thought, there have always been those that tried to understand the human being through the human body. Thus, the field of biological psychology evolved.
Today, there is a wide range of knowledge that is accepted as fact in the study of biology. The central nervous system is the controlling agent in bodily behaviour. It is composed of the brain and the spinal cord and its basic unit is the neuron. The neuron communicates with other neurons using action potentials and neurotransmitters (Carlson, 2004).
Neurotransmitters from the synaptic vesicle of the terminal button of a neuron can cause a graded potential. Unlike action potentials which are always depolarization, graded potentials may be either depolarization (excitatory) or hyperpolarization (inhibitory) (Kalat, 2007). Depending on whether the neurotransmitter hyperpolarizes or depolarizes the neuron, there are two basic postsynaptic potentials that take place in the human body – Inhibitory postsynaptic potential (IPSP) and excitatory postsynaptic potential (EPSP).
During EPSP, the sodium channels are opened and sodium ions enter the cell and but that from a single synapse is not enough to reach the threshold of excitation. Thus, the EPSP decays over time and space. An IPSP on the other hand involves the opening of potassium channels allowing the potassium from inside the cell to leave and also for the negatively charged chloride ions to enter. Thus, the cell



References: Bagley, E. E. et. al. (2011). Drug-induced GABA transporter currents enhance GABA release to induce opioid withdrawal behaviors. Nature Neuroscience, 14(12), 1548-1554. doi: 10.1038/nn.2940 Carlson, N.R. (2004). Physiology of Behaviour (8th ed.). Boston: Allyn & Bacon. Comai, S., Tau, M. & Gobbi, G. (2012). The psychopharmacology of aggressive behavior: A translational approach: Part 1: Neurobiology. Journal of Clinical Psychopharmacology, 32(1), 83-94. doi: 10.1097/JCP.0b013e31823f8770 Halgin, R. P. & Krauss, S. W. (2011). Abnormal Psychology: Clinical Perspectives on Psychological Disorders. New Delhi: McGraw Hill Kalat, J. W. (2007). Biological Psychology (9th ed.). Belmont, CA: Thompson Wadsworth. Kurchavyi, G. G., Kalinina, N. I. & Veselkin, N. P. (2012). Contributions of glycine and GABA A receptors to the generation of inhibitory postsynaptic potentials in frog spinal cord motoneurons. Neuroscience and Behavioral Physiology, 42(1). doi: 10.1007/s11055-011-9537-z Steiger, H., Bruce, K. R. & Groleau, P. (2011) Neural circuits, neurotransmitters, and behavior: Serotonin and temperament in bulimic syndromes. In Adan, R. A. H. & Kaye, W. H. (Eds.), Behavioral Neurobiology of Eating Disorders (pp. 125-138). New York, NY, US: Springer-Verlag Publishing Storozheva, Z. I. & Proshin, A. T. (2011). Selective involvement of the neurotransmitter systems of the cerebellum in the mechanisms forming different types of defensive behavior. Neuroscience and Behavioral Physiology, 41(9), 964-972. doi: 10.1007/s11055-011-9516-4

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