Lipid Digestion Of Lipids Lab Report

Question #1 Pre-load can be defined as the degree to which cardiac (heart) muscle cells extend before contraction in the heart. Pre-load is one of the factors that affect the stroke volume. It directly relates to stroke volume. The way it works is that a rise in venous return to the heart raises the end diastolic volume of the ventricle which causes the muscles fibers to stretch thus increasing preload. Also, it increases the force of contraction of the ventricles making the heart to discharge the returned blood further. Hence, an increase in EDV leads to an increase in SV and vice versa. Contractility can be defined as the contractile strength achieved at a given muscle length. In contractility, end diastolic volume (EDV) is the primary intrinsic component controlling stroke volume. However, the external elements that expand heart contractility can also boost SV. The more calcium in a cell
Digestion of lipid does not occur in the mouth and stomach because enzymes that would aid the digestion are not present there. The major organ for the digestion of lipids in the small intestine because the pancreas is the primary source of lipid-digesting enzymes (lipase). For the body to digest the fat molecules in the food, a process must occur, and it is called “Emulsification (ability to mix with water).” This is because triglycerides are insoluble in water (cannot be mixed with water). The bile is responsible for emulsification; it also can mix fat and water and attach to the fat molecule to make it more soluble. In the duodenum, the bile splits big lipid globules into minute fat droplets so that lipase enzymes can have enough surface area to function. After emulsification, the pancreatic lipase which is the major enzyme for lipid digestion begins by first breaking off two of the fatty acid chains which would allow monoglycerides and free fatty acids; this begins the process of