Ovarian Lesions Essay

The ovarian lesions is one of the three female genital lesions in the world, and its mortality rate ranks the first in gynecological oncology. It was reported that the risk factors for ovarian cancer include infertility, late menopause, early menarche and so on. The protective factors have multiple pregnancy, breastfeeding and oral contraceptives. Recently, a link among platelet count and hyperfibrinogenemia and malignancies, such as gastric, lung, esophageal, colorectal, pancreatic, cervical, ovarian, and endometrial cancer was revealed. Thrombocytosis was also observed in some patients with cancer. In the present study we synchronously investigated the clinical significances of plasma fibrinogen and platelet count in ovarian lesion. With the deterioration of ovarian disease, the
Platelets are from the megakaryocytes produced by bone marrow hematopoietic stem cells, and they are not only the smallest component of nuclear-free cells in blood circulation, but have the adhesion, aggregation, secretion, and blood clots procoagulant systolic functions. The platelets are indicators of cells production and decline, and the platelet count is more common in clinical laboratory testing. The definition of polyplastocytosis has no uniform standard in ovarian lesions. Some studies define the peripheral platelet count [22], but there are other different standards expect for this, for example Iwasaki et al. [23] define the cut-point of platelet count as . Soonthornthum et al. [24] studied cases of ovarian cancer Thailand patients, which takes the platelet count as the standard. Although the standard of polyplastocytosis has not reached an unified standards, but most scholars at home and abroad defined the value of polyplastocytosis as . In our study, we use the platelet count as the polyplastocytosis, which is consistent with the