nerves, or psychological stress attributed to the traumatic event caused phantom limb syndrome to occur (Scott, 2011). Current research and theory focuses on brain plasticity and self organization processes.
However, phantoms are most commonly reported after amputation of an arm or leg there have been reports following the amputation of parts of the face, breast, or even internal viscera (McCormick et al., 2013), for example, an individual can have sensations of bowel movement after removal of the sigmoid colon and rectum. Previous reports have found some paraplegics have had phantom erections and ejaculations, and reports of women having phantom menstrual cramps after a hysterectomy. McCormick et al., (2013), states “sensory memories can reemerge in the phantom in spite of deafferentation”. Deafferenatation is the loss of the sensory input from a portion of the body, usually caused by interruption the peripheral sensory fiber such as, an amputation of a limb.
The internal representation the body is controlled in the brain is created by feedback mechanism of skin and muscle sensory afferents (Scott, 2011).
The sensations are not illusions––these sensations occur because the brain relays motor signals to the missing body part, but does not receive sensory feedback. The sensations are perceived as movements as if the limb still exists, even though the individual can visually recognize the limb is absence. The brain simply unable to interpret the conflicting sensory information experienced by the amputee (Scott, 2011). Thus, requiring experience and time for the brain to adjust to the loss of a limb, because the brain retains information from the imaged of an intact body
part. In original claims children born without limbs were the obvious exception to the theory posed by Mitchell. These individuals have never received sensory signal to emphasize the idea of a body image in the brain.