Research Paper On Erectile Dysfunction

Erectile Dysfunction an Enemy of the Sexes Erectile Dysfunction is a disease that plagues greater than 30 million men and their partners in the United states, with as few as 6 million seeking professional help and even less getting medical treatment. What is Erectile dysfunction, and what is the pharma-co-mechanical ups and downs associated with the issue? The questions still loom as to the enemies that bring on ED. Can ED be prevented or is this an inevitable downfall to life and its endless disparities of aging and disease. What’s the science behind the treatment, is it affective, can more be done? Is this just the tip of the iceberg causing a meltdown of male and female pleasure and reproductive properties, ultimately raising havoc to the
The physiology of an erection is the relaxation of the cavernous smooth muscle of the penis. Corpora cavernosa, two chambers that that are contained in the penis made of “spongy tissues when the spaces between the tissue are filled the penis becomes erect, when the veins constrict this aids in the longevity of the erection,” (Adams, Holland & Urban, 2014), which loses its zeal with the outflowing of the blood. The parasympathetic response is in relation to the olfactory, visual and tactile stimuli, neurotransmitters are released that relax the cavernous smooth muscle. “Increased nitric oxide (NO) being the primary conductor for an erection, inhibiting Rho kinase, assisted by acetylcholine (ACH) and vasoactive intestinal polypeptide (VIP), stimulating a fall in cytosolic calcium (Ca+2),” (Shamloul & Ghanem, 2013; Steers, 2002) if Ca+2 is increased the penis loses its ability to remain erect. In its normal mechanism this happens automatically with a complete orgasm and ejaculation, but with dysfunction this loss of erection occurs prematurely before the pleasure, is ever