Preview

Tired Swimmer

Satisfactory Essays
Open Document
Open Document
353 Words
Grammar
Grammar
Plagiarism
Plagiarism
Writing
Writing
Score
Score
Tired Swimmer
Shelby Giles
June 22, 2015
The Tired Swimmer

1. She is experiencing constant fatigue, blurred vision, eye strain, gasping for air, hand and finger aches.

2. Blurred vision and eyes strain could have to do with the optic nerve, headache, soreness and fatigue.

3. In the spinal cord there are nerves that are in charge of these involuntary functions. There is also the medullary rhythmicity centers in the medulla oblongata that adjusts the rate of breathing. These could be possibilities that Annies is having problems breathing.

4. My guess is that she is experiencing something wrong with her central nervous system. Her signs and symptoms are indicative of multiple sclerosis or another neurological disease.

5. More eyestrain, double vision/blurry vision, stinging eyes and droopy eyelids.

6. A decline in muscle function can arise from miscommunication at the neuromuscular junction resulting in dysfunction due to neurons not firings to produce contraction of the eye muscles and cilliary muscle.

7. Annie had the conduction test done and the electrodes that simulated the muscle had no problem with nerve fatigue and the conduction velocity is fine. Annies nerve function is normal.

8. The first test results showed that her muscles were normal, the second test showed that her “muscle response decreased quickly during repeated nerve stimulation.”

9. Her skeletal muscles fatigued during repetitive stimulation due to possible failure along the somatic motor pathway involved in muscle activity from the CNS.

10. Sodium & potassium
Sodium ions vesticular membrane neurotransmitter e) vesicles
f) closes
g) action potential
h) presynaptic membrane
i) neurotransmitter is taken up

11. The Thymus plays a key role in training and development of T-Cells/T-Lymphocytes.

12. A toxin or other foreign substance that induces an immune response in the body.

13. Annies antigens will attack the antibodies, she will feel no muscle movement or action potential. The

You May Also Find These Documents Helpful

  • Good Essays

    If a motor neuron is damaged, it can cause the muscle it innervates to atrophy because of lack of stimulation.…

    • 10962 Words
    • 62 Pages
    Good Essays
  • Good Essays

    R. V. Latimer Case Brief

    • 579 Words
    • 3 Pages

    palsy, Because of this she required around the clock care by her parents. She was…

    • 579 Words
    • 3 Pages
    Good Essays
  • Good Essays

    Case Study 4

    • 1348 Words
    • 5 Pages

    Because motor neurons degenerate with ALS, they can no longer send impulses to the muscle fibers that normally result in muscle movement. Early symptoms of ALS often include increasing muscle weakness, especially involving the arms and legs, speech, swallowing or breathing. When muscles no longer receive the messages from the motor neurons that they require to function, the muscles begin to become smaller. Therefore limbs begin to look "thinner" as muscle tissue atrophies.…

    • 1348 Words
    • 5 Pages
    Good Essays
  • Good Essays

    The tremors that Annette is experiencing could also be related to the primary motor are of the brain because the basal ganglia of this area also control muscle tone and help to suppress unwanted movements.…

    • 432 Words
    • 2 Pages
    Good Essays
  • Powerful Essays

    PHYSICAL EXAMINATION: Revealed a pleasant but depressed appearing female who is over weight but in no acute distress. She wears a brace on her right wrist. Height is 5 feet 7 inches. Weight 252 pounds. Temperature 98°F. Pulse 80. Respiratory rate 20. Blood pressure 140/80. She has not carotid bruits or meningismus. Cranial nerves 2 through 12 are intact and detailed to include visual fields. Funduscopic exam and pupillary examination. Motor exam reveals 5 out of 5 strength in arms and legs without atrophy or vesiculation. Reflexes are trace over four. Sensory exam is negative and nonfocal.…

    • 502 Words
    • 3 Pages
    Powerful Essays
  • Satisfactory Essays

    13. Assume the surgery to reconnect blood vessels was successful and blood flow was re-established immediately throughout the arm. Would the nerves of the arm recover as quickly and completely as the muscles? Explain why or why not.…

    • 253 Words
    • 2 Pages
    Satisfactory Essays
  • Satisfactory Essays

    She then told the doctor that she had a baby about 5 months ago. Then the doctor told her since it has been about five days you are a lucky young lady. I don’t believe this is a stroke, “I think that this is Bell’s palsy. I then told her not to worry it was still in the primary stages and that I would need to put her on a steroid and a antibiotic to help her get better. I then began to tell her that she was lucky she came when she did because some people stay with Bell’s palsy without ever getting it corrected.…

    • 728 Words
    • 2 Pages
    Satisfactory Essays
  • Satisfactory Essays

    lesson 4 case study

    • 289 Words
    • 1 Page

    Not getting essential fatty acids because not enough lipids are being consumed and lacking protein. No nourishment to muscles. Causing her to be weak and tired…

    • 289 Words
    • 1 Page
    Satisfactory Essays
  • Good Essays

    Annette s Story

    • 333 Words
    • 1 Page

    C) What other symptoms that Annette is having is also controlled by this region of the brain?…

    • 333 Words
    • 1 Page
    Good Essays
  • Powerful Essays

    The Tired Swimmer Case

    • 613 Words
    • 3 Pages

    3. How could a decline in muscle function contribute to eye strain and blurred vision?…

    • 613 Words
    • 3 Pages
    Powerful Essays
  • Satisfactory Essays

    Epilepsy Case Criteria

    • 467 Words
    • 2 Pages

    Her vital signs are normal. Weight is 170 pounds. The HEENT exam shows her fundi are benign. Pupils and EOMS are intact. She has a fairly large linear…

    • 467 Words
    • 2 Pages
    Satisfactory Essays
  • Better Essays

    Myasthenia Gravis

    • 1358 Words
    • 6 Pages

    Myasthenia gravis is a chronic, progressive neuromuscular, autoimmune disease marked by varying degrees of weakness of the skeletal (voluntary) muscles of the body. The body’s immune system attacks and destroys receptors in the muscles. These receptors bind acetylcholine, the neurotransmitter released from motor neurons. (Turkington & Harris, 2009) The main calling card of myasthenia gravis is muscle weakness that increases during periods of activity and improves after periods of rest. Muscles that control eye and eyelid movements, facial expression, chewing, talking, and swallowing are often, but not always, involved. The muscles that control breathing and neck and limb movements may also be affected. (Beers, 2003)…

    • 1358 Words
    • 6 Pages
    Better Essays
  • Good Essays

    notices that the numbness and tingling not only persists, but has also spread up to her knees. Again, she ignores the abnormal sensation. By the end of a month’s time, the numbness spreads to the midline of her body. At this point, she becomes alarmed.…

    • 1458 Words
    • 6 Pages
    Good Essays
  • Satisfactory Essays

    a. The binding of ACh opens ion channels in the dendrites or cell body that…

    • 278 Words
    • 2 Pages
    Satisfactory Essays
  • Good Essays

    imbalances or tissue damage, hence the loss of the ability to contract, or muscle fatigue.…

    • 589 Words
    • 3 Pages
    Good Essays