Design process 1. We are making an insulin receptor model showing how the receptor works on the cell and allows glucose flow though it. 2. We need to make a model out of some kind of craft material that will allow for us to portray a insulin receptor and will move easily for demonstration 3. – insulin receptors are mainly on the outside of the cell and have multiple components to them * Main job of an insulin receptor is to allow for the insulin to bond to the receptor to
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Report on Receptor Proteins According to Abedon (2012)‚ receptor proteins are intracellular proteins‚ or protein fractions‚ that have a high specific affinity for binding a known stimulus to cellular activity. Receptor proteins may be located at cell surfaces or within the interior of cells. They may detect hormones or instead keep track of what otherwise is going on in cell ’s environment. They may be enzymes or serve to relay the occurrence of signal reception in other ways such as by opening
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Cell Size Control David A Guertin‚ Whitehead Institute for Biomedical Research‚ Cambridge‚ Massachusetts‚ USA David M Sabatini‚ Whitehead Institute for Biomedical Research‚ Cambridge‚ Massachusetts‚ USA Growth in biological systems is defined as the accumulation of mass‚ which leads to an increase in size. In this article‚ we discuss how cells‚ organs‚ and organisms normally control growth‚ and how deregulated growth can lead to a variety of pathological conditions. Introductory article Article
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Physiology Linder Unit 1 Possible Essay Questions Chapter 1 1. Describe the concept of homeostasis. Explain how negative feedback and positive feedback loops effect the homeostatic condition. Use examples to explain your answer. Homeostasis can be described as maintaining a relatively internal environment even though the external environment is variable. Negative feedback loops effect the homeostatic condition by controlling it by bringing you back to your set point
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Rafa Mwais Professor Hermann Chem 157 Paper #5 May 28‚2013 Crystal Structure of the Catalytic Portion of Human HMG-CoA Reductase 3-hydroxy-3methylglutaryl-CoA reductase (HMGR) catalyzes the formation of mevalonate in the biosynthesis of sterols and isoprenoids. The activity of this enzyme is regulated physiologically through synthesis‚ degradation and phosphorylation to maintain to maintain the concentration of the mevalonate-derived products. Transcription and translation of HMGR increase
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The effect of alcohol concentration on the cell membrane Abstract: In this experiment I found out that as the concentration of the alcohol was increased so did the colour intensity of the solution. This is because the higher concentration of ethanol‚ results in more damage done to the cell membrane‚ resulting in leakage of red pigment from the cell. If the membrane is damaged more‚ more red pigment will leak out of the membrane and into the ethanol. Hypothesis: The higher the concentration
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Urine production If body fluids are hypo osmolar‚ then kidney will produces hypo osmolar urine. (Dilute urine) If body fluids are hyper osmolar (dehydration) then kidney produces hyperosmolar urine. (concentrated urine) In the renal cortex the osmolarity of interstitial fluid is 300mOs/L going to 1200mOs/L in the papilla. From the cortex to the papilla there is an increasing hyper osmolarity of the interstitial fluid. Cortico papillary osmolarity gradient. Urine with osmolarity more than 300mOs/l
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Chapter 4: Membrane Structure and Function Fluid Mosaic Model 1. Consistency of membrane is solid enough to form a boundary‚ but flexible and permeable like a fluid. 2. Proteins scattered throughout - Embedded or on the surface - Can move around laterally 3. Phospholipid from bilayer‚ make up most of membrane 4. Glycolipids have carbohydrate chains attached. 5. Hydrophilic heads point out/into the cell 6. Hydrophobic tails point out towards each other Proteins 1. Glycoproteins: have
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INTRODUCTION Both scarring and nonscarring alopecias have been described with long-term use of epidermal growth factor receptor (EGFR) inhibitors. Current treatments revolve around anti-inflammatory agents such as topical corticosteroid ointments‚ creams‚ and intralesional injections to reduce follicular inflammation. Unfortunately‚ none of these therapies are universally effective. Here‚ we describe a case of erlotinib-induced alopecia refractory to intralesional and topical corticosteroids that
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Botulinum neurotoxins (BoNTs) are the most potent toxins known and the causative agent of botulism. Their exceptional toxicity is caused by the highly efficient inhibition of neurotransmitter release at the neuromuscular junction. High affinity binding of BoNTs is mediated by the simultaneous interaction with lipid embedded synaptic vesicle proteins and gangliosides the presynaptic membrane according to the well-established dual-receptor binding model. One peculiarity that is especially pronounced
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