Nikul Patel DS13 25-12-03 Effects of exercise on cardiac output (For results see graph paper) Analysis After the initial period of rest in both girls and boys heart rates rose slightly with light exercise (walking)‚ and then fell again slightly back towards the original resting point. Then during the period of anticipation of exercise the males heart rate rose by a further 1 beat per 6 seconds‚ whereas the females took a dramatic leap‚ and rose by 3.5 beats per 6 seconds‚ or 35 beats per minute
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PH210 PHARMACY CLERKSHIP HOSPITAL PUTRAJAYA Case 5 Congestive Cardiac Failure Nephrotic Syndrome Community-Acquired pneumonia Siti Hasmah Bt Mohd Suffian (2006200606) CASE SUBJECT Name: MAM Gender: Male Age: 24 years old Race: Malay Weight: 138 kg BMI: 55 (morbidly obese) Height: 159cm DOA: 21st March 2010 SUBJECTIVE Chief complaint: • bilateral lower limb swelling for the past 2 weeks. • scrotal swelling for the past 3 days but noted on DOA had progressively
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Cardiovascular Disease Ashley Cookie Cardiac Arrest Mr. Stapleton November 20th‚ 2012 Would you like to feel or be close to death? How would you like to have your heart pump rapidly out of control? A cardiac Arrest is the absence of ventricular contraction that results in systemic circulatory failure. A Cardiac Arrest is something that can happen out of nowhere without you knowing. You can be cooking and you just collapse and go into Cardiac Arrest. It’s that easy! And many people
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the use of an Automatic External Defibrillator (AED). According to the Heart Rhythm Society “An estimated 382‚800 people experience sudden cardiac arrest (SCA) in the United States each year” (Sudden Cardiac Arrest Facts). Only 32% SCA victims get CPR because most bystanders do not have training. Training CPR and AED would save lives (About SCA: Sudden Cardiac Arrest: A Healthcare Crisis). b. Thesis statement: Making CPR and AED training a graduation requirement would make students able to help
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Heart Failure ➢ Renin-Angiotensin System Blockers ➢ (-Blocker ➢ Diuretics ➢ Direct Vasodilators ➢ Inotropic Agents ➢ Aldosterone Antagonists 4) Drug Treatment of Cardiac arrhythmias ➢ Class I (Sodium Channel Blockers) ➢ Class II ((-adrenoreceptor Blockers) ➢ Class III (Potassium Channel Blockers) ➢ Class IV (Calcium Channel Blockers) Antihypertensive Drugs: Diuretics:-
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Social Responsibility & Diversity (EGYPT’S KING OF HEARTS) Introduction As a general manager at the Central Auditing Organization (C.A.O); the supreme Auditing firm responsible for auditing ministries‚ public institutions‚ non for profit organizations and any other entity assigned to the C.A.O by the high authorities‚ I found In
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(Hint: relate your response to the refractory period of the cardiac action potential) How well did the results compare with your prediction? The threshold doesn’t change because after the voltage was simulated the cardiac muscle cells Ca channels opened. You cannot open them anymore or reopen them before depolarization. This is due to the refractory period. 3) Why is it only possible to induce an extrasystole during relaxation? Because cardiac muscle is able to depolarize after repolarization takes
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saline on cardiac output? HYPOTHESIS: Cardiac output is higher in a hot saline and low in cold saline temperature parameters. Educated Guess Answer: Phenomenally cardiac output is supposed to rise in hot saline conditions and drastically go down in cold saline conditions. Normalising Data Table 2.1: Cardiac output normalised data of frogs in normal‚ hot and cold saline conditions Normal Hot Cold FROG SV(mV/beat) HR(beat/min) Cardiac output(L/min) SV(mV/beat) HR(beat/min) Cardiac output(L/min)
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Sample Physiology MCQs 1. Compound action potentials can be graded responses. True 2. The sciatic nerve contains axons of both afferent and efferent neurones. True 3. The absolute refractory period of an action potential is the period after an initial stimulus when it is impossible to evoke another action potential with a second stimulus. True 4. The refractory periods of individual axons are important as they dictate the maximal action potential firing rate of a neuron. True 5. Compound action
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Practice development issues around Induced Therapeutic Hypothermia (ITH) on Cardiac Arrest Patients Contents 1. Introduction 2. Pathophysiological changes during cardiac arrest and return of spontaneous circulation 3. Physiological benefits of therapeutic hypothermia 4. Guideline for induced therapeutic hypothermia after cardiac arrest 5. The UHL guideline’s goal of therapeutic hypothermia 6. Preparation‚ Monitoring and Supportive therapy 7. Cooling Methods 8. The relative experience 9
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