patient showed presenting symptoms of a respiratory distress (SPO2 of 89%)‚ fever (102.4°F)‚ weight loss‚ productive cough‚ dyspnea‚ crackles and wheezing in lungs‚ pleural effusion‚ and malaise. S.S. has several co-morbid factors affecting his recovery: COPD‚ Diabetes Mellitus Type II‚ hypertension‚ chronic renal insufficiency‚ depression‚ and history of benign prostatic hyperplasia (BPH). The patient had a transurethral resection of the prostate (TURP) two years to treat the BPH. Recent surgical history
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and management of chronic obstructive pulmonary disease (COPD) in an acutely ill patient. The acutely ill patient involved in this essay was admitted to hospital due to cerebrovascular accident and had a past medical history of myocardial Infarction‚ left Ventricular failure‚ peripheral vascular disease and duodenal ulcer as well as chronic obstructive pulmonary disease. This essay will provide a rationale for the chosen aspect of care (COPD) and reason will be given why it is a priority. In particular
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R.S. is a long-time smoker who developed bronchitic chronic obstructive pulmonary disease (COPD). He also has a history of coronary artery disease and peripheral vascular disease. His arterial blood gas (ABG) values are pH = 7.32‚ PaCO2 = 60 mm Hg‚ PaO2 = 50 mm Hg‚ HCO3 - = 30 mEq/L. His hematocrit is 52% with normal red cell indices. He is taking an inhaled ß agonist and theophylline to manage his respiratory condition. At his clinic visit‚ it is noted that R.S. has an area of consolidation in his
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Analysis [clinical complexities] ………………………………… 2 2.1 Complex Issues [COPD and hypertension] ………………… 2 2.2 Complex Issues [Prednisolone use] ………………………….. 2 Chapter 3 Prednisolone Pharmacokinetics …….………………………………… 3 Chapter 4 Prednisolone Pharmacodynamics …….……………………………… 4 Chapter 5 Nursing Considerations 5.1 Nursing considerations [COPD] ..…………………………….. 5 5.1.1 Clinical Manifestations of COPD .…………………….... 6 5.2 Nursing considerations [prednisolone use]
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1. What clinical findings are likely in R.S. as a consequence of his COPD? - COPD‚ chronic obstructive pulmonary disease is mostly consists of two main diseases: emphysema and chronic bronchitis. A patient with COPD can show various clinical findings which can include dyspnea‚ cyanosis‚ wheezing‚ “Blue bloaters” clubbing of the finger nails‚ and pink puffers. Also‚ the patient can show continuation of productive chronic coughing with expectoration within last 3 months for two consecutive years
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also concentrating on the many systems that can be affected as a result of the illness or disease. In the patient with Chronic Obstructive Pulmonary Disease (COPD)‚ the care of the patient offers challenges in management‚ and is greatly improved by an open systems approach to patient care. In order to analyze the origin‚ pathophysiology‚ and effects of the disease‚ an open system allowing many components to
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(ACE) inhibitors? Where is the ACE located? When are most prescribed? What medical diagnosis warrants an ACE inhibitor? How do they work? While defining the remainder o/the vocabulary words‚ ask the following questions: Angioedema: Describe the pathophysiology behind Angioedema. How are ACE inhibitors related to Angioedema? What is the treatment for Angioedema? What is the treatment for ACE~ inhibitor-induced Angioedema? Answer: Angioedema is a localized edema involving the deep‚ subcutaneous layers
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by: Miguel‚ Stefani Gil M. Case RP is a 72 year-old male who was admitted to the hospital from his long-term care facility after 1 week of dyspnea and cough. He was seen by a staff physician at the longterm care facility and was diagnosed with a COPD(Asthma) exacerbation. He has been suffering of Osteoarthritis since he was at the age of 56. Consequently‚ He is now taking steroid drugs to alleviate the pain from the disease. Furthermore‚ He smokes a lot too. Review of systems reveals fever‚ chills
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Henry Williams is a 74-year-old African American who was admitted for shortness of breath secondary to chronic obstructive pulmonary disease exacerbation. His past medical history is chronic obstructive pulmonary disease (COPD)‚ cardiovascular disease‚ hyperlipidemia‚ asthma‚ hear loss‚ and hypertension. His neighbor brought him to the emergency room because he was having difficulty breathing and weakness. When he was brought to the emergency room‚ he was very weak and restlessness. Williams breathing
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Cardiovascular systemE227 Group 1-Aetiology of disease in Scotland & Health Models and Health Promotion – Mike Keenan | ThursPON20.09.2012F124 | Intro to the module BarbaraKilloran | Pathophysiology of atherosclerosis‚ PVD & MI Group 1Tamsin MacBride | | Intro to the moduleBarbara Killoran | Pathophysiology of atherosclerosis‚ PVD & MI Group 2Tamsin MacBride | | | Group 2 STUDY | | | Group 1 STUDY | FridayPON21.09.12F124 | Assessment & principles of care of a patient with CVS
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