Clinical Manifestations of endocarditis – mostly non-specific |Table 124–2 Clinical and Laboratory Features of Infective Endocarditis | | | |Feature
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Information about infective endocarditis is important for all nurses to have a basic understanding. Although rare‚ it can affect anyone at any time and health care personnel should be educated about the manifestations and outcomes it can have on people. Without knowing about the disease process‚ it can be hard to communicate with a family about what their loved one may be going through. Patients and family members will ask questions‚ and nurses need to be prepared and educated to answer truthfully
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Introduction Endocarditis is an infection of the inner layer of the heart (endocardium) or an infection of the heart valves. Endocarditis can cause growths inside the heart or on the heart valves. Over time these growths can destroy heart tissue and cause heart failure or problems with heart rhythm. They can also cause stroke if they break away and form a blood clot in the brain. Early treatment offers the best chance for curing endocarditis and preventing complications. What are the causes? This
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Introduction Clinically defined‚ Endocarditis is an infection of the inner lining of the heart. Endocarditis occurs when bacteria enters the body and spreads through the blood stream. Once in the blood‚ the bacteria will latch on to any damaged or weakened parts of the heart‚ and in most cases the heart valves are targeted. Endocarditis is extremely rare for individuals with healthy hearts and immune systems‚ but those with weakened immune systems‚ preexisting heart conditions‚ or individuals that
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temperature. Bacterial endocarditis is an infection of the inner surface of the heart or the heart valves caused by bacteria usually found in the mouth‚ intestinal tract or urinary tract. This infection results in a serious illness which requires prolonged treatment and on occasion produces injury to the heart or even death. Endocarditis is a major concern in almost all unrepaired congenital heart defects as well as in most repaired defects with a few exceptions. Endocarditis is characterized by a
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Case Study 15 Scenario J.F. is a 50-year-old married homemaker with a genetic autoimmune defi ciency; she has suffered from recurrent bacterial endocarditis. The most recent episodes were a Staphylococcus aureus infection of the mitral valve 16 months ago and a Streptococcus mutans infection of the aortic valve 1 month ago. During this latter hospitalization‚ an ECG showed moderate aortic stenosis‚ moderate aortic insuffi - ciency‚ chronic valvular vegetations‚ and moderate left atrial enlargement
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Infective endocarditis is a type of endocarditis which is classified as a cardiac disorder. It affects the endocardium which is the inner most layer of the heart‚ targeting mainly the valves but may also affect the heart chambers (Kumar‚ Abbas‚ Aster‚ 2015). This disease has an infective component which means pathogens have entered the area and disrupted the normal tissue (Cotran‚ Kumar‚ & Collins‚ 1999). Infective endocarditis may lead to severe valvular insufficiency‚ which inturn could possible
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Background: Systemic embolism is a common complication of infective endocarditis‚ most frequently involving the central nervous system‚ spleen‚ kidney‚ liver‚ and iliac or mesenteric arteries but embolisation to coronary artery causing sudden cardiac death is infrequently encountered. Case presentation: A case of 45 years old male who had coiling for anterior communicating
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the diagnosis of infective endocarditis. Ariane had her tongue and nose pierced 6 weeks ago. The drug screen is negative. She presents with tricuspid insufficiency murmur grade II‚ and a temperature of 104°F. The patient complains of extreme fatigue‚ and anorexia. The echocardiogram reveals vegetations on the tricuspid valve. What risk factors predisposed Ms. Waters to develop infective endocarditis? Body piercings can be major risk factors for infected endocarditis‚ especially the tongue piercing
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maculopapular to pustular‚ often with a hemorrhagic component. Lesions are peripherally located‚ and may be painful before they are visible. Fever is common but rarely exceeds 39°C. Rare complications of DGI include gonococcal meningitis‚ pericarditis‚ and endocarditis. Headache‚ neck pain and stiffness‚ fever‚ and decreased sensorium may indicate gonococcal meningitis. This disease may be clinically indistinguishable from meningococcal meningitis on presentation‚ although the course of gonococcal meningitis
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