known. We have studied the effects of fever and convulsions on water and electrolyte balance in CSF and serum by measuring osmolality and electrolyte concentrations in children. The febrile population consisted of 60 children‚ 36 of whom had seizures during fever. Twenty-one children without convulsions and nine children with epileptic symptoms were nonfebrile controls. We noticed that CSF is subject to changes in osmolality and electrolyte concentration during fever‚ while convulsions do not exhibit
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all people HEAT IS PRODUCED BY: • Metabolism • Increased muscle activity • Vasoconstriction • External sources HEAT IS LOST BY: • Vasodilation • Convection • Radiation • Conduction • Evaporization TEMP or FEVER? • TEMPERATURE—the measurement of heat in the body • FEVER—the measurement of heat in the body
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the cytomegalovirus (CMV)‚ which is also a herpes virus. Both EBV and CMV have a tendency to become latent in host cells. Incubation periods are often lengthy (4-8 weeks). Symptoms include extreme fatigue‚ sore throat‚ high fever‚ weight loss‚ and often pharyngeal inflammation. Symptoms usually persist for 2-3 weeks‚ occasionally even longer. Fatigue symptoms often persist even longer. Infections are often likely to be permanent‚ but is almost never fatal. Some
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SECTION ONE INTRODUCTION AND NOTIFICATION 1. INTRODUCTION AND NOTIFICATION 1.0 INTRODUCTION The guidance is divided into sections as follows: Section 1 Introduces infection control and explains notification; Section 2 deals with general infection control procedures; Section 3 gives guidance on the management of outbreaks; Section 4 describes specific infectious diseases; Section 5 contact numbers and sources of information; Section
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diplococcus causes pneumonia‚ OM‚bacteremia & meningigits. D. Erythema Infectiousum ____Whooping cough; paroxysmal cough‚ esp nocturnal. E. HIB ____Epstein-Barr virus; herpes group. Accompanied by hepatosplenomegaly‚ Fever‚ tonsillitis‚ lymphadenopathy. F. Lyme Disease ____Fifth’s Disease; slapped cheeks. Not contagious after the rash appears; Assoc. with fetal demise with exposure in first trimester. G. Rubeola ____Erythema Migrans; often in the groin
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Perspectives of the death of alexander the great On june 10th 323 bc alexander the great died . The actual cause of his death remains unknown to this day. There are many different perspectives on the death of alexander the great died. Some say he was murdered and some say he died of natural causes but from evidence from his diary entry’s there are very few perspectives that could be right. There two perspective’s that I believe are the closest too the truth is other that he died from being
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Diseases Rabies Hantavirus Trichinosis Mosquito-borne Diseases Protozoa Helminthiasis Virus Tick-borne Diseases Colorado Tick Fever Rocky Mountain Spotted Fever (Tick-borne Typhus) Lyme Disease Tularemia Relapsing Fever Other Tick-borne Diseases Flea-borne Diseases Plague Murine Typhus Fever Commensal Rodent-borne Diseases Rat-bite Fever Leptospirosis (Weil’s Disease) Salmonellosis Rickettsialpox Bird-borne Diseases Histoplasmosis Ornithosis (Chlamydia psittaci
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CSEC BIOLOGY SUMMER RESEARCH PAPER DISEASE & ITS IMPACT ON HUMANS This research is aimed at informing the reader on different types of diseases and the impact it has on humans. Enclosed herein are details on the types‚ treatments‚ transmissions and descriptions of different diseases in the region and around the world.
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endotoxin) Lipoopolysaccharides means (lipid (A) and sugar-(0-antigensaccharides) and the core antigen is variable Once u give antibiotic the condition get worse because endotoxin release cause (inflammation) so in pedriatic pretreat with steroid ( board exam) Destroying outer membrane will first release Lipid A causing pt to get worse before getting better -Severe shock Gram negative sepsis. pediatrics- give steroids before antibiotics for bacterial meningitis (dexamethasone BEFORE antibiotics) to
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Alexander Fleming Biography… Fleming was born on 6 August 1881 at Lochfield a farm near Darvel in East Ayrshire Scotland. After the war Fleming actively searched for anti-bacterial agents‚ having witnessed the death of many soldiers from septicemia resulting from infected wounds. Unfortunately antiseptics killed the patients’ immunological defences more effectively than they killed the invading bacteria. In an article he submitted for the medical journal The Lancet during World War I‚ Fleming described
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