Questionnaire Design Which brand of shampoo do you use? - Taken from ‘Marketing Research’ by Dr. Naresh K. Malhotra • “Who” – Respondent uses personally or the brand used by the household? • “What” – If more than one brand of shampoo is used? Should the respondent mention most preferred brand or recently used brand or the brand used most often or the brand that comes to the mind first? • “When” – Does the researcher means last time‚ last week‚ last month? • “Where” – Although it implied that
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Just how sweet is artificial sweeteners? Statistically this sugar imposter is thirty to eight thousand times sweeter than real sugar‚ but at what cost? All artificial sweeteners were discovered in a lab‚ and the substance the scientists discovered just happened to be sweet. Artificial sweeteners were never intended to be sweeteners. Pure‚ raw sugar is much better for your body than anything that was founded in a lab. Aspartame is derived from GMOs and is found in diet soda‚ but recently pepsi has
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Research indicates that there is a positive correlation between consumption of high fructose corn syrup and obesity percentages. Sucrose is the leading added sweetener in in the manufacture of foods in the Unites States and is the biggest source of fructose. Sucrose and high fructose corn syrup (HFCS) are metabolized identically and react the same way to insulin‚ leptin and ghrelin. Sucrose is indifferent from HFCS in causing obesity. Compared to glucose‚ sucrose is extracted directly from the liver
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concluded that fructose‚ a sugar that found in fruits and honey‚ is much worse for us than glucose that found in bread‚ rice‚ and potatoes (Brefere & Drummond‚ 2017‚ p. 68). Even though they are both single sugars‚ they have different effects on our body. The same as people questioned for many years whether the calories are the same in cookies as in salads‚ right not we trying to understand the difference in carbohydrates. Since modern diet of American consists of lots of fructose‚ question rises
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High Fructose Corn Syrup and Obesity High fructose corn syrup (HFCS) is one of the major caloric sweeteners used in the food industry today. It is a sweetener that contain fructose and all provide 4 kcal per gram (Suzan‚ 620). It is relatively inexpensive to produce‚ so the food industry sees HFCS an easy way to make products less expensive. HFCS is a kind of corn sugar; it is nearly identical to sucrose (table sugar)‚ which is made up of 50 percent glucose and 50 percent fructose (“What”)
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reaction because the enzyme stays the same‚ it does not change during the reaction. (Hudon-Miller‚ Enzymes‚ 2013) Enzymes act as protein catalysts in biochemical processes Frutokinase is the enzyme that acts on fructose in the liver and turns fructose into fructose 1 phosphate. Fructose 1 phosphate is changed to DHAP and glyaraldehyde by an enzyme called aldolase B.
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Methods: An observation was made on whether or not the sugars‚ fructose and ribose would be fermented in yeast (Saccharomyces cerevisiae). By looking at the chemical structures of these sugars given in (Figure 1)‚ we looked for anything that might determine the metabolization of these sugars. An equal 6mL (milliliter) sample of 2.5% fructose and yeast suspension was pipetted into a fermentation tube. The same procedure above was performed with the sugar ribose which has a concentration of 2
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1 Western Governors University 2 Hereditary Fructose Intolerance Metabolism is the process of cellular respiration. It involves three steps‚ 1) glycolysis‚ 2) the citric acid or Krebs cycle and 3) electron transport system. The glycolytic pathway or glycolysis is a metabolic process that leads to the formation of the energy source adenosine triphosphate (ATP) in the body. ATP is essential to the cell and the cellular processes used by the cell. The first step‚ glycolysis is the process
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together in the proper position and make them to react each other. Deficiency in aldolase B and hereditary fructose intolerance * Hereditary fructose intolerance is a condition I which affects a humans ability to digest the fructose sugar. The incidence of hereditary fructose intolerance is 1to 2 in 20000 to 30000 individuals in a year worldwide. .( John .R.H 1996) * Hereditary fructose intolerance can be caused by mutations in the ALDOB gene. The ALDOB gene is responsible for making the aldolase
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available substrate. Hereditary Fructose Intolerance AldolaseB is found mainly in the liver. The second step in fructose metabolism is where aldolaseB breaks down fructose-1-phosphate into glyceraldehyde and dihydroxyacetone phosphate. If there is a deficiency in aldolaseB‚ it slows down the metabolism of fructose allowing for a buildup in the liver. A toxic build up of fructose in the liver can cause the death of liver cells. Hereditary Fructose Intolerance If fructose-1-phosphate is not being
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