How to Quickly and Easily Understand Your Blood Tests Without A Medical Degree The Simplified Patient Reference Guide By Ronald J. Grisanti D.C. Limits of Liability/Disclaimer of Warranty The author‚ Ronald Grisanti and publisher‚ Busatti Corporation have made their best effort to produce a high quality‚ informative and helpful book. The author and Publisher make no representation or warranties with respect to the accuracy‚ applicability‚ fitness or completeness of the contents of this program
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Topic 1: Subject‚ tasks and methods of pathophysiology. General nosology‚ etiology and pathogenesis. 1. A 49-year-old man was suffering 12 years ago from rheumatic myocarditis‚ endocarditis‚ and insufficiency of mitral valve. Examinations showed the absence of inflammatory process‚ sufficient minute blood volume. What is it? A. Pathological reaction B. Pathological process C. Typical pathological process D. Compensatory reaction E. Pathological
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Individual Purification Report The 60% saturation of ammonium sulfate was performed to precipitate out LDH and other proteins. Components such as nucleic acids and sugars‚ which are more soluble‚ remained in the supernatant. In this step 0.13g of ammonium sulfate salt was slowly added per each ml of the 40% supernatant as the solution was stirring. Enzyme assay and protein assay were performed. The results indicated 4600±100 unit enzyme activity concentration and 75±2 mg of protein in the 60% pellet
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Alcohol related liver diseases are a wide range area which refers to a general term named as alcoholic liver diseases. Millions of individuals around the world get affected with liver diseases due to high alcohol consumption. Several risk factors have been analysed that affects the risk of development and evolution of liver disease. The primitiveness and estimation of alcohol related alcoholic liver diseases depends on the pattern and term of alcohol consumption‚ presence of liver inflammation‚ diet
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| Immunisation program Age due | IMMUNISATION | No. of injection | BIRTH | BCG | 1 | 2months | 6in1 and PCV:Diphtheria/Tetanus/Whooping Cough(Pertussis)/Hib(haemophi- lus influenza B)/IPV(Inaetivated Polio)/Hepatitis B (6in1)‚Pneumococcal conjugate vaccine (PCV) | 2 | 4months | 6in1 and Men C: Diphtheria/Tetanus/ Whooping Cough/Hib/Polio/Hepatitis B (6in1)‚ Meningococcal (Men C) | 2 | 6months | 6in1 and PCV and Men
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The metabolic response to prolonged starvation Early stage of prolonged starvation Starvation occurs when the body has a severe lack of nutrients needed to survive. The body has ways of adapting to periods without food‚ for example overnight it needs to survive without any additional nutrients whilst asleep. The body however also has ways of adapting for as long as long as 5-6 weeks (Garber‚ 1974). Therefore‚ looking at the metabolic state during starvation is over a long time period. The body
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become more sexually exploring. Men and women drink equally‚ however‚ women can become drunk much faster than men. A few reasons are: women are typically smaller than men. Women have less body fat and less of an enzyme called‚ alcohol dehydrogenase. Alcohol Dehydrogenase is found in the human stomach and allows alcohol to be broken down in the body. The enzyme is more prevalent in men than in women‚ causing the effects of alcohol to be felt much faster. But most people know when there limit is reached
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Treatment of Fetal Alcohol Syndrome Like any condition‚ treatment for fetal alcohol syndrome is constantly emerging. Since the syndrome causes neurological changes similar to attention deficit hyperactivity disorder it was once thought that medications used to treat attention deficit hyperactivity disorder would be useful in treating fetal alcohol syndrome.2 Conflicting results from the use of the medications challenged doctors to conduct treatment differently. Pharmacists and doctors are now looking
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enzyme is bound to the enzyme know further reaction can take place until it dissociates from the molecule hence it is often termed a dead end complex (Palmer‚ 1991). A classic example is the competitive inhibition of succinate dehydrogenase by malonate‚ succinate dehydrogenase catalyses the oxidation of succinate to fumarate. Another example is the inhibition of sulphur drugs that inhibit the synthesis of sulphur drugs. Competitive inhibition for enzymes that have a single substrate and single binding
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Makati Medical Center College of Nursing DRUG STUDY Patient’s Name: M.D.C. Age: 42 y/o Medical Diagnosis: Leptospirosis Attending Physician/s: M.T Allergies: none Allergic Responses: none Prepared by: Shiela Guiquing |DRUG NAME |MECHANISM OF ACTION |RATIONALE FOR THIS PATIENT |ADVERSE DRUG EFFECT |NURSING IMPLICATIONS | |
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