"Metabolic acidosis and alkalosis" Essays and Research Papers

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    Muscle Fatigue
By Jaclyn Mullick B.S. The onset of muscle fatigue has hindered many athletes from achieving their maximum performance. Over the years we have been taught that it is the build-up of lactic acid that is the downfall of muscle fatigue and soreness. This is only partially true; there is a little more to it than what we have been told. I hope that this article provides you with a possible short-term remedy and does not confuse you more but rather makes you more aware

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    General Adaption Syndrome- (Stress Response) Three Stages: Consistent way the body reacts to stress; typically involves elevated levels of epinephrine and glucocorticoids Alarm reaction- mediated by norepinephrine from the sympathetic nervous system‚ and epi from the adrenal medulla. They prepare body for flight or fight. Angiotensin and aldosterone levels also increase. Angiotensin raises BP‚ aldosterone promotes sodium and H2O conservation‚ which helps offset possible losses by sweating and bleeding

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    vo2 max

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    to pump out blood‚ the ability of the tissues to extract oxygen from the blood‚ the ability to ventilate and the ability of the alveoli to extract oxygen from the air. At rest‚ nearly all of the body’s energy demands are being met by aerobic metabolic processes‚ which require oxygen. The mitochondria are the site of aerobic metabolism in the cells (aerobic metabolism will be covered in greater detail in labs later this quarter). Ultimately‚ oxygen is the final electron acceptor in the electron

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    contract from these three choices water‚ Gatorade‚ and Powerade. The role of water in our body are giving cell life‚ where water is a carrier‚ distributing essential nutrients to cells as minerals‚ vitamins‚ and glucose. It also has chemical and metabolic reactions as it removes waste products including toxins that the organ cells reject and removes through urine and feces. Water is a transport of nutrients in the biochemical break down of what we eat. H2O regulates body temperature‚ it has a large

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    Super Germ Research Paper

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    Some call it “horror” and some call it “the super germ”‚ but now‚ our always known “regular” bacteria‚ those one-celled creatures once considered under control with antibiotics‚ have invaded our hospitals and headlines with a vengeance. The vengeance used against us is caused by an existing organism called necrotizing fasciitis‚ the so-called flesh-eating bacteria‚ caused by Group A streptococcus. What this organism does is progressively destroy the human body

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    COPD Case Study

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    Pathophysiology Dr. Isabelle Tardif August 7‚ 2015 Abstract The case study presents the outcome of smoking that resulted to RS’ chronic bronchitis and chronic obstructive pulmonary disease. His ABGs’ show partially compensated respiratory acidosis as manifested by decreased pH‚ increased PaCO2‚ decreased PaO2 and increased HCO3. RS most likely has the following clinical findings caused by COPD: enlarged right heart along with the signs and symptoms of the right-sided failure‚ secondary polycythemia

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    Adrenal Gland

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    Adrenal gland In mammals‚ the adrenal glands (also known as suprarenal glands) are the triangular-shaped endocrine glands that sit on top of the kidneys. They are chiefly responsible for releasing hormones in conjunction with stress through the synthesis of corticosteroids and catecholamines‚ including cortisol and adrenaline (epinephrine)‚ respectively. Anatomy and function Anatomically‚ the adrenal glands are located in the retroperitoneum situated atop the kidneys‚ one on each side. They are

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    Septic Shock

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    pressure may be decreased or increased depending upon fluid resuscitation Compromised nutrient blood flow to organs; decreased organ oxygen extraction ● PATHOPHYSIOLOGYCont. Pulmonary & Renal ● ● ● ● ● ● ● Hyperventilation with respiratory alkalosis Pulmonary hypertension and edema Hypoxemia (arterial pO2 < 50 mmHg) Reduced pulmonary compliance; increased work Respiratory muscle failure Renal hypoperfusion; oliguria Acute tubular necrosis and renal failure PATHOPHYSIOLOGYCont. Other ●

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    17 - Fetal Surveillance (11) Acceleration: Reassuring - Description/Appearance: o Rate increase of at least 15 beats/minute that lasts for a minimum of 15 seconds o Rate increases often in response to fetal movement o May accompany contractions - Cause: o Interaction between sympathetic and parasympathetic nervous systems - Nursing Interventions: o None Early Decelerations: Reassuring - Description/Appearance: o Rate decrease during contraction o Often seen in late labor when head

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    A&P Notes Ch. 1-3

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    Chapter 1 Section 1 Anatomy-Study of internal and external structures of the body and the physical relationships among body parts Gross Anatomy -involves the examination of relatively large structures and features usually visible with the unaided eye Microscopic Anatomy-deals with structures that cannot be seen without magnification Physiology-the study of function and considers the functions of the human body Anatomy and physiology are closely interrelated Theroretically Practically Anatomical

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