"Monoclonal antibodies" Essays and Research Papers

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    is a very unique and important person in the medical world. He is one of the most punctual donors alive‚ and has changed the world for many. James Harrison is an extremely rare and matchless case. He is one of the few people alive that have the antibodies for Rh Incompatibility in their blood‚ or other wise plasma. There are only fifty other people know in Australia to have this rarity flowing in their blood (Bresnahan). Harrison has broken world records for donating. He has saved two-point-four

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    Dugo

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    that consist of: * 91% Water * 7% Protein * 2% Others (Ions‚nutrients) Plasma contains dissolved proteins. * Albumin – makes of 58% of the plasma protein. Important contribution in Osmotic Pressure. * Globulin – 38%. These are antibodies; has clotting factors. * Fibrinogen – 4%. Activation of clotting factors converts fibrinogen to fibrin‚ a threadlike protein that forms blood clots. Serum is plasma w/o clotting factors. Formed Elements * Red blood cells or erythrocytes

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    pharmaceuticals

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    1.Intro: A pharmaceutical drug (medicine or medication and officially medicinal product) is any chemical substance formulated or compounded as single active ingredient or in combination of other pharmacologically active substance‚ it may be in a separate but packed in a single unit pack as combination product intended for internal‚ or external or for use in the medical diagnosis‚ cure‚ treatment‚ or prevention of disease. 2. History: The development of small molecule therapeutic agents for

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    Myasthenia Gravis

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    Myasthenia gravis Overview: Myasthenia Gravis is an autoimmune disorder of the post-synaptic neuromuscular junction whose pathogenesis consists in an attack on antibody-mediated At the acetylcholine receptor site on the postsynaptic membrane of the neuromuscular junction. Causes: Characteristics of the disease are muscle weakness and fatigue. The pattern of symptoms is typically fluctuating; it is more pronounced at night and improves with rest. Initial symptoms include: ptosis‚ diplopia or blurred

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    Systemic Lupus Case Study

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    Case Study A 17-year-old female with systemic lupus A 17-year-old female was admitted with a month long history of involuntary movement bilaterally in hand and legs. The patient stated that the movement started one month ago. And she had occasionally involuntary movement of her mouth. The symptoms were mild in the beginning and gradually increased. The patient had dyskinesia around her mouth and thoracic and occasionally choreathetoid movement in bilateral upper extremity. She said she had similar

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    Tuberculosis is a common‚ and in many cases lethal‚ infectious disease caused by various strains of mycobacteria‚ usually Mycobacterium tuberculosis. Tuberculosis typically attacks the lungs‚ but can also affect other parts of the body. It is spread through the air when people who have an active TB infection cough‚ sneeze‚ or otherwise transmit respiratory fluids through the air. Most infections are asymptomatic and latent‚ but about one in ten latent infections eventually progresses to active disease

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    Cells come from pre-existing cells via division Cells of all organisms fundamentally similar in structure‚ function‚ and metabolic functions Contain hereditary information passed down during cell division A cell’s volume increases faster than it’s surface area Less surface area = less are for nutrient/waste diffusion Solves via: Dividing‚ getting thinner‚ form microvilli‚ store nutrients (Microvilli – within small intestine) Cells are highly organized‚ 3 basic parts Plasma membrane Selective

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    prednisone and reportedly has a couple of calls into his office and has not had a return call back. She is not sure how she should proceed regarding that‚ at this time. The patient has also been to see Dr. Catcher‚ a hematologist because of a monoclonal

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    Second Line Of Defense

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    group of chemicals called lymphokines. Some lymphokines kill the pathogens directly‚ others stimulate lymphocytes to become active and help increase the inflammation so that there are more macrophages. Helper T-cells co-operate with B-cells in the antibody production. They also activate macrophages and promote inflammation. Memory cells remain even after the pathogens have been killed to stop

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    the frequency of thyroid antibodies and ATD is similar to that in classical T1DM. This findings suggests that T1DM co-morbidities may be common in clinical T2DM patients who have B-cell autoimmunity. Despite their obesity‚ youth with insulin requiring diabetes should be screened for thyroid and possible other T1DM associated autoimmune

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