channelopathy characterized by muscle weakness or paralysis with a matching fall in potassium levels in the blood (primarily due to defect in a voltage-gated calcium channel). In individuals with this mutation‚ attacks often begin in adolescence and are triggered by strenuous exercise followed by rest‚ high carbohydrate meals‚ meals with high sodium content‚ sudden changes in temperature‚ and even excitement‚ noise or flashing lights. Weakness may be mild and limited to certain muscle groups‚ or more severe
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Polymyositis Polymyositis is an autoimmune disease that affects several muscles in your body. It can be classified as a persistent inflammatory muscle disease that causes weakness of the skeletal muscles‚ which controls movement in the body. In severe cases polymysitis can affect major organs like heart‚ and lungs. The symptoms of Polymyositis come with extreme pain‚ marked with weakness and loss of muscle mass closest to the truck particularly those in your thighs‚ shoulder‚ upper arm‚ and
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number of muscle diseases that weaken the musculoskeletal system and hampers movement according to Yeole‚ Gurunani‚ Dhole and Yogesh (2014). Emery (2002) states these disorders produce muscle wasting and weakness of variable distribution and severity. Muscular dystrophies are caused by genetic mutations that have a progressive degenerative effect on an individual’s muscles stated Punnoose‚ Burke and Golub (2011). The muscles most often affected include limbs‚ facial muscles‚ respiratory muscles‚ cardiac
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what is muscular dystrophy The muscular dystrophies are a group a group of genetic diseases characterized by progressive weakness and degeneration of the skeletal or voluntary muscles which control movement. The muscles of the heart and some other involuntary muscles are also affected in some forms of muscular dystrophy‚ and a few forms involve other organs as well. History of muscular dystrophy Children’s Hospital Boston has a long history of conducting innovative research on muscular dystrophy
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dry mouth‚ and muscle weakness clearly illustrate a correlation between botulism and the muscular system. Synaptic transmission at a motor end plate normally begins when a nerve impulse reaches a synaptic terminal. Synaptic vesicles then travel towards and combine into the presynaptic cell membrane of the motor neuron. This stimulates the release of acetylcholine‚ which diffuses across the synaptic cleft and binds to receptors on the postsynaptic cell membrane of a muscle fiber. When a botulin
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Muscular Dystrophy‚ also referred to as “MD”‚ is a group of diseases that cause progressive weakness and loss of muscle mass (Mayo). In MD‚ abnormal genes interfere with the production of proteins needed to form the muscle’s health (Mayo). There are many long-term effects to this terrifying disease. The primary one‚ that being unable to walk. Muscular Dystrophy is a life-threatening disease that many pose questions and desire to learn more about. Muscular Dystrophy occurs in various forms. While
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Briefly‚ Anatomically‚ the skin and muscles of the forearm ‚ hand and fingers are innervated by three major nerves‚ which are; the Median‚ the Radial and the Ulnar nerves. These nerves can be susceptible to injuries and compression at different locations on their paths as they innervate the skin and muscles of the forearm‚ hand and fingers. Having established that‚ the carpal tunnel is a relatively narrow passageway that is formed anteriorly by the flexor retinaculum and posteriorly by the carpal
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called muscular dystrophies. It is the most common form of muscular dystrophy that begins in adulthood. “Myotonic dystrophy is characterized by progressive muscle wasting and weakness” (Myotonic‚ 2010). typically people who have this disorder have whats called myotonia‚ a prolonged muscle contraction‚ and they are not able to relax some muscles. For example‚ someone with myotonic dystrophy may have difficulty releasing their grip on a doorknob or handle. Sometimes‚ they have slurred speech or locking
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like | | | |curare‚ onto the cholinergic receptor found on the | | | |skeletal muscle fibers | |-Toxins |κ-Toxin |Blocks some of the central nervous system’s cholinergic | | |
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communication. Your brain ordered nerve cells‚ to activate muscles in your hand and fingers. In this instance‚ your muscles responded‚ and your fingers moved. In a person afflicted with Amyotrophic Lateral Sclerosis (ALS)‚ there is a break in the chain of communication. The motor neurons degenerate and die. Because of the lack of motor neurons‚ the brain cannot communicate with the muscles‚ and voluntary muscle movement is no longer possible. Because the muscles are no longer used‚ they too begin to degenerate
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