fungi‚ and bacteria. Viruses that infect bacteria are called bacteriophages‚ or phages. The nucleic acid of a virus is protected by a protein coat called the capsid. Each capsid is composed of protein subunits called capsomeres. In some viruses‚ the capsid is covered by an envelope‚ which usually consists of some combination of lipids‚ proteins‚ and carbohydrates. Depending on the virus‚ envelopes may or may not be covered by spikes‚ which are carbohydrate-protein complexes that project from the
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The virus known as Measles There are many viruses today that at one time were a deadly disease‚ however because of advances in technology and vaccinations‚ we no longer give them a second thought. Prior to the discovery of a vaccination‚ the United States reported a total of over 50‚000 cases of measles outbreaks each year with 450-500 cases resulting in death. (NCIRD) Since the disbursement of an effective vaccination‚ the measles has been almost eradicated in the United States; however it is
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what’s the difference between a bacteria and a virus. A bacterium is one of the three living domains of life. It is a prokaryote which means it does not house a nucleus or membrane-bounded organelles. However‚ it does have a nucleoid region where the DNA is stored. It can be said that bacteria is more like us because they can share their DNA with one another through conjugation and even pick up other DNA through transformation. On the other hand‚ a virus is not made up of cells. They are acellular.
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More commonly known “kennel cough” virus‚ it can be easily spread through contact at dog parks and grooming facilities. Bordetella bronchiseptica is a highly contagious bacterium causing a virus that affects a large number of dogs across all parts of the globe. Tracheobronchitis is brought on through bacteria that affect the dog’s upper respiratory system. After exposure‚ clinical signs of Bordetella bronchiseptica can been seen within 2-14 days with the virus lasting normally from 14-20 days. The
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Chapter 33 notes: Intrinsic cellular defense against virus infection 1. Toll- like receptors sense virus infections a. Toll-like receptors (TRL) i. Located on the cell surface or in endosomal membranes = recognize conserved structures found in pathogens ii. Highly conserved receptors from flies humans iii. Most mammals have between 10-15 (humans = 10) iv. TLR – 2: lipoproteins/glycoproteins (mealsels‚ hepB‚ herpes) v. TLR– 3: dsRNA (west
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animallike and funguslike. A virus is a microscopic particles that make up either DNA or RNA core covered by a protein coat. Viruses are so small an electrn microscope is needed to see them. Viruses are not classified in any kingdom because they are not cells. They show almost none of the characteristics of living things. The classification of viruses are based on it’s shape‚ the kind of nucleic acid it contains‚ and the type of organism the virus infects. The protein coat of a virus gives the particle it’s
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Varicella zoster virus is a double stranded icosahedral‚ nucleocapsid DNA molecule‚ with a lipid envelope enclosing it. Varicella zoster virus develops a latent infection in the neurons of the sensory ganglia. Primary infection causes varicella (chickenpox)‚ and reactivation of the virus causes a latent infection known as herpes zoster (shingles). Varicella is more common in children. Infection in adults is more severe and can lead to interstitial pneumonia. It is also more frequent in immunocompromised
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A bacteriophage (informally‚ phage) is a virus that infects and replicates within bacteria. The term is derived from ’bacteria’ and the Greek φαγεῖν phagein "to devour". Bacteriophages are composed of proteins that encapsulate a DNA or RNA genome‚ and may have relatively simple or elaborate structures. Their genomes may encode as few as four genes‚ and as many as hundreds of genes. Phage replicate within bacteria following the injection of their genome into the cytoplasm. Bacteriophage are among
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Modeling the Course of a Viral Illness and its Treatment Math Internal Assessment Student: Xu‚Dejing(Charlotte) Session number:001762023 Date: March 26‚ 2010 I. Introduction: When viral particles of a certain virus enter the human body‚ they replicate rapidly. In about four hours‚ the number of viral particles has doubled. The immune system does not respond until there are about 1 million viral particles in the body. The first response of the immune system is fever. The rise in temperature
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factors govern the severity of JEV pathogenesis. The failure of the host to produce antibodies against the virus is associated with an increased likelihood of the disease to turn lethal (Ghosh D‚ 2009). An important factor in the increase of pathogenesis is crossing the blood –brain barrier and this result in neurotropic viral infection. After entering the body through a mosquito bite‚ the virus reaches the central nervous system (CNS) via leukocytes where JEV virions bind to the endothelial surface
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