Sickle Cell Anemia Pathophysiology: Sickle cell anemia is a disease passed down through families in which red blood cells form an abnormal crescent shape. The “sickling” occurs because of a mutation in the hemoglobin gene. Sickle cells are stiff and sticky. They tend to block blood flow in the blood vessels of the limbs and organs. Blocked blood flow can cause pain‚ serious infections‚ and organ damage. Life expectancy in people who have this disease is shortened. Anemia is a condition in
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ineffective rhythm and ineffective emptying causes decreased cardiac output and stasis of blood. The stasis that results can cause harmful clots to pool in the atria putting patients at risk for thromboembolic stroke‚ pulmonary embolus‚ or renal embolus. An estimated 15% to 25% of strokes are associated with AF (Hardin & Steele‚ 2008). Predisposing Factors Several factors place patients at risk for AF. A history of hypertension and/or coronary heart disease seems to be the most common underlying
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Cushing’s syndrome. Firstly‚ according to a clinical study‚ Cushing’s syndrome is a chronic excessive exposure of glucocorticoids of the body’s tissues of the high level of the hormone cortisol‚ also known as hypercortisolism. In normal adrenal pathophysiology‚ the production of hormone cortisol produced within adrenal gland by the zona fasciculata of the adrenal cortex. Adrenal glands located in the superior
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Examples of leading causes of death in Australia for 2005 are ischaemic heart disease and lung cancer‚ knowledge of this information allows us to prioritize how much funding goes into preventative measures and curative measures of ischaemic heart disease and lung cancer Life expectancy is one measure used to determine health priority areas. Life expectancy is the projected average number of years
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days. Cardiologist at UCSD recommends Cardiac Thrombectomy to prevent further strokes. Neurologist recommends endovascular intervention to prevent future embolic strokes though not during an acute episode. Patient was held at UCSD ED for permissive hypertension during acute stroke. Patient complained of cough with green phlegm over the past few days; chest x-ray findings of no local infiltrate. Pathophysiology:
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Related Disorders. (2nd ed.). Baltimore‚ William and Wilkins‚ p. 55-68 Gemmell‚ H Herzog‚ W. (2002). Clinical Biomechanics of Spinal Manipulation. New York‚ Churchill Livingston‚ p. 92-190 Hong‚ C Hong‚ C.Z. (2000). “Myofascial trigger points: pathophysiology and correlation with acupuncture points‚” Acupuncture in Medicine‚ vol. 18‚ no. 1‚ p. 41-47 Imamura‚ S.T.‚ Fischer‚ A.A.‚ Imamura‚ M.‚ Teixeira‚ M‚J.‚ Tchia‚ Yeng Lin‚ Kaziyama‚ H.S.‚ et al Lavelle ‚ E.D.‚ Lavelle‚ W. & Susti‚ H.S. (2007)
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MITRAL VALVE DISEASE MITRAL STENOSIS Mitral stenosis is almost always rheumatic in origin‚ although in the elderly it can be caused by heavy calcification of the mitral valve apparatus. There is also a rare form of congenital mitral stenosis. In rheumatic mitral stenosis‚ the mitral valve orifice is slowly diminished by progressive fibrosis‚ calcification of the valve leaflets‚ and fusion of the cusps and subvalvular apparatus. The flow of blood from left atrium to left ventricle
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Copstead‚ L.-E Dementia With Lewy Bodies Information Page. (2011‚ December 28). Retrieved March 8‚ 2012‚ from National Institute of Neurological Disorders and Stroke: http://www.ninds.nih.gov Dugdale‚ I Frontotemporal Dementia Information Page. (2011‚ December 28). Retrieved March 8‚ 2012‚ from National Institute of Neurological Disorders and Stroke: http://www.ninds.nih.gov Gillick‚ M Lexicomp. (2012). Retrieved February 5‚ 2012‚ from Lexicomp: https://online-lexi.com Midthun‚ M Parkinson’s disease.
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TUTORIAL 5 HEART FAILURE Pathophysiology B. Left-sided HF SOB A. Right-sided HF Orthopnea Edema in the feet‚ ankles‚ legs‚ liver‚ & abdomen. If the amount of fluid is fluid accumulates in the abdomen. Fluid accumulation in the liver or stomach nausea & loss of appetite. Eventually‚ food is not absorbed well loss of weight & muscle. This condition is called Cardiac Cachexia Paroxysmal nocturnal dyspnea Tiredness & weakness A sudden accumulation of a high amount
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following assignment I will be reviewing the evidence relating to the use of milrinone‚ a phosphodiesterase inhibitor that is commonly used to increase cardiac output in children after cardiac surgery. I will focus on LCOS in children and the pathophysiology surrounding this. I will also examine the biological action of milrinone and how it has been shown to increase cardiac output in children after
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