20 TYPICAL ANGINA PECTORIS 20 ANTI ISCHEMIC AGENTS 20 PREVENTIVE AGENTS 20 VASODILATORS: 20 CALCIUM CHANNEL BLOCKERS: 22 ADRENOCEPTOR ANTAGONISTS: 25 MANAGEMENT OF ANGINA PECRORIS: 26 DRUGS FOR HEART FAILURE: 27 OVERVIEW: 27 PATHOPHYSIOLOGY OF HEART FAILURE: 28 MECHANISM AND EFFECT OF DRUG FOR HEART FAILURE: 29 ANTIHYPERTENSIVE DRUGS: OVERVIEW: A n estimated 50 million people in the united states have high blood pressure (hypertension) ‚ commonly defined as a sustained systolic
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had a Myocardial Infarction at the age of 40. His cholesterol level is 250 mg/dl‚ HDL is 30 mg/dl‚ LDL is 170mg/dl and TG is 250mg/dl. Mr. S complains of chest pain upon exertion. Case Study # 1 Questions 1. Briefly describe the pathophysiology of CAD (25 pts) 2. Calculate Mr. S’s BMI (25pts) 3. Based on all the evidence do you
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Cited: Danbolt‚ N. (2001). Glutamate as a Neuro Transmitter. The Neurotrasporter Group‚ 100-105. Gorelick‚ P. (1986). Alcohol and Stroke. current Conceptes of Cardiovascular Disease‚ 268-271. McCance‚ K.‚ & Huether‚ S. (2006). Pathophysiology: The Biological Basis for Disease in Adults and Children. Moseby Inc. Osacar-Berman‚ M.‚ & Bowirrat‚ A. (2005). Genetic influnces in emotional dysfunction and alcoholism-related brain damage
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Table of Contents Introduction ..3 Risk Factors .3 Pathophysiology ...4 Clinical manifestation ..5 Diagnostic criteria 5 Laboratory and Diagnostic test 6 Evaluation & Treatment ...6 Prognosis ..6 Summary ..7 There
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Childhood Obesity America’s childhood obesity rate is at an all time high. Many adolescents are failing the standard for healthy eating habits‚ which is the reason for their obesity. As you may know‚ an excessive amount of saturated fat in a diet will lead to weight gain. The amount of overweight children aged 6 to 11 years has nearly tripled in the past 30 years‚ going from 7% to a shocking 18.8% (Krisberg). Obese children often struggle with physiological troubles‚ such as low self-esteem and
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Doctor’s Order h. Social History i. Family History IV. Physical Assessment 7 j. Vital Signs upon admission V. Anatomy & Physiology 9 k. Heart l. Myocardium VI. Pathophysiology 12 m. Pathophysiology Chart n. Four Stages VII. Laboratory Results and Diagnostic Procedures 16 o. Arterial Blood Gas Analysis p. Bacteriology Report q. Chemistry Section r. Chest X-Ray s
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CFP 208 A1 Nursing Challenges in Care Imagine waking up in the morning and no knowing your partner or spouse lying next to you in the bed. Imagine waking up and not knowing your own name‚ how old you are or when your birthday is. Imagine having to look at your children‚ grandchildren‚ brothers and sisters and asking who they are. Imagine seeing the one you devoted your life to and them not remembering you. Imagine going to visit them every day and every time having to explain to them who you are
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INTRODUCTION Hypertensive disorders of pregnancy‚ previously known as Pregnancy Induced Hypertension (PIH)‚ are high blood pressure disorders of pregnancy. It has long been one of the major problems for mothers in pregnancy‚ along with infection and postpartum hemorrhage. PIH is classified as gestational hypertension‚ mild preeclampsia‚ severe preeclampsia and eclampsia‚ depending on how far development advances. A woman is said to be preeclamptic when her blood pressure increases and shows protein
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IHCD Paramedic Module H Assessment Using the answer sheets provided‚ encircle your answer against the corresponding question number. 1 Weight has the greatest effect on the amount of kinetic energy produced a. TRUE b. FALSE 2 Blunt trauma produces temporary cavitation a. TRUE b. FALSE 3 Sympathetic action causes pupil dilation a. TRUE b. FALSE 4 A capillary refill of more than 4 seconds indicates serious circulatory compromise a. TRUE b. FALSE 5 In time critical patients‚
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myocardial contractility‚ heart rate As heart rate increases‚ this allows less time for diastole and the heart often cannot fill adequately. Therefore‚ a very rapid heart will often have a reduced cardiac output‚ especially in an elderly individual Stroke Volume Dependent on PRELOAD Amount of blood ejected by the ventricle with each contraction The volume of blood prior to the contraction (end diastolic vol) – the volume blood in the ventricle at the end of the contraction (end systolic vol) = SV Applies
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