are called the atria and the bottom chambers‚ the ventricles. A normal heart beat begins when the Sino-Atrial Node‚ located on the posterior wall of the right atrium. The Sino-Atrial Node initiates the "wave of contraction" which results in the contraction of the atria. The Atrio-Ventricular Node delays the signal initiates by the Sino-Atrial node for about 0.15 seconds‚ permitting full atrial systole before ventricular systole. This ensures that the ventricles are filled before the ventricles contract
Premium Heart
two chamber Pre Test • 3) A breast cancer patient has significant shortness of breath symptoms and echo reveals a moderate sized pericardial effusion. Which of the following echo features does not support tamponade physiology? – – – – A) Right atrial systolic collapse B) Right ventricular diastolic collapse C) Inferior vena cava collapse D) Marked respiratory variation of tricuspid valve inflow Pre Test • 4) The following echo features are noted in a patient with shortness of breath and dyspnea
Premium Heart Cardiology Medical imaging
infarction. • Normal sinus rhythm starts in the sinoatrial (SA) node and follows the normal conduction pattern of the cardiac cycle. o The P wave represents the depolarization of the atria (passage of an electrical impulse through the atria)‚ causing atrial contraction. o The PR interval represents the time period for the impulse to spread through the atria‚ atrioventricular (AV) node‚ bundle of His‚ and Purkinje fibers. o The QRS complex represents depolarization of the ventricles (ventricular contraction)
Premium Cardiac electrophysiology Cardiology Myocardial infarction
electrical conduction disturbances? A. Atrial fibrillation B. Ventricular fibrillation C. Ectopic foci D. Premature ventricular contraction 3. A common adverse effect from quinidine overdosage is A. Hypertension B. Tachypnea C. Cinchonism D. Dry mouth 4. Disopyramide produces a _____ in conduction and _____ of the refractory period. A. Increase‚ contraction B. Increase‚ prolongation C. Decrease‚ contraction D. Decrease‚ prolongation 5. In atrial flutter‚ one would expect the ECG
Free Cardiology Heart Atrial fibrillation
comes into the cardiology office‚ complaining of fatigue‚ palpitations‚ shortness of breath with mild activity‚ ongoing consistently for the past week without any chest pain or syncopal episodes. She is a 75-year-old woman with a history of atrial fibrillation (AFib)‚ controlled type 2 diabetes‚ myocardial infarction (MI) with a history of percutaneous coronary intervention to right coronary artery‚ stage IV chronic renal failure‚ and a mild mitral regurgitation on ECHO 7 years ago. Her in clinic
Premium Patient Health care Nursing
What are the characteristics of ventricular fibrillation? 2. Describe the changes in atrial pressure‚ ventricular pressure‚ aortic pressure and ventricular volume that occur during the various stages of the cardiac cycle. Illustrate when the various valves are open or closed. 3. In order for both of the semilunar valves
Premium Heart Cardiology Blood
Most of the cardiac injury ultimately results in development of cardiac fibrosis due to negligible regenerative capacity of heart. Cardiac fibrosis is associated with increased deposition of matrix proteins in the myocardium. The expansion of the cardiac interstitial space in absence of significant cardiomyocyte loss is “reactive interstitial fibrosis” and the formation of scar in response to myocardial infarction is called “reparative fibrosis” [10‚ 46]. Increased deposition of endomysial and perimysial
Premium Heart Myocardial infarction Blood
Health Teaching A Paper Presented to Rima Nepangue RN‚ MSN‚ College of Nursing Adventist University of the Philippines In Partial Fulfillment Of the Requirements for the Course Advance Health Assessment By Aris Calangi HEALTH TEACHING This is the profession of educating people about health. Areas within this profession encompass environmental health‚ physical health‚ social health‚ emotional health‚ intellectual health‚ and spiritual health. It can be defined as the principle
Premium Stroke Myocardial infarction Health
render care for the patient‚ the student will be able to: * Discuss the overview of the disease in the review of the Related Literature. * Identify its clinical manifestation. * Present the Anatomy and Physiology * Trace the pathophysiology of the disease * Establish a good and therapeutic nurse-patient interaction. * Monitor patient’s progress. * Determine the status of the patient through: a. General Data b. Physical
Premium Stroke
inferior-Posterior infarction The above ECG changes can be found if there is an occlusion of the: RCA LAD circumflex all of the above. the most complications associated with this problem is ventricular dysrhythmias. AV block. atrial flutter. hemodynamic changes and dysrhythmia caused by SA node & AV node. Identify the Rhythm in the below figure Unifocal Bigeminy. Multifocal trigeminy Unifocal Trigeminy Multifocal PVCs. The nurse would be most concerned
Premium Myocardial infarction