NEUROLOGICAL SYSTEM DIVISIONS OF THE NERVOUS SYSTEM: 1. Central Nervous System (CNS)=Brain & Spinal Cord 2. Peripheral Nervous System (PNS) a. Autonomic Nervous System=System Homeostasis b. Sympathetic Nervous System=Fight or Flight c. Parasympathetic Nervous System=Rest & Digest d. Enteric Nervous System=GI system NEURONS 1. 3 parts of a nerve cell a. Dendrites b. Cell Body c. Axon 2. Types of nerves: a. Afferent Neurons=sensory‚ conduct impulses TO the brain b. Efferent Neurons=motor
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cholinergic nerves. Acetylcholine is found in both the peripheral nervous system and the central nervous system. Acetylcholine can also be found in the autonomic nervous system and is the only neurotransmitter used in motor division of the somatic nervous system. In the peripheral nervous system acetylcholine plays a role in skeletal muscle movement‚ as well as in the regulation of smooth muscle and cardiac muscle. In the central nervous system acetylcholine is believed to be involved in the sleep-wake
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-Morphine- agonist Difference with Somatic and Autonomic nervous system (Peripheral ) Somatic Nervous System Made up of nerves that connect to voluntary skeletal muscles and to sensory receptors. -Consists of neurons that communicate between the body and the brain Peripheral Nervous System Made up of all those nerves that lie outside the brain and spinal cord. Difference between afferent and efferent neurons (Somatic Nervous System) Efferent Neurons -Motor neurons -Neurons that carry messages
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Guide – Nervous System Part II 1. What are the 2 major divisions of the Nervous System? Central Nervous System and Peripheral Nervous System 2. What are the 2 major divisions of the central nervous system (CNS)? The brain and the spinal cord 3. What are the 2 major divisions of the peripheral nervous system (PNS)? The somatic nervous system and autonomic nervous system. 4. Differentiate between the autonomic and somatic nervous systems. The somatic nervous system is a body
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pain‚ relates to nerve injury or dysfunction of the nervous system. This essay is going to explain mechanisms of central and peripheral neuropathic pain. It will describe causes and symptoms and provide some examples of common neuropathic syndromes. This piece will also discuss ways of treatment‚ including pharmacological and non-pharmacological methods. 2 CAUSES Neuropathic pain originates from pathology of the nervous system due to alterations in neural input or processing
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arms/hands and legs/feet‚ causing sensory loss in the skin and muscle weakness. This may cause the person with leprosy to lose an extremity affected by the disease. B: Schwann cells are involved in many important aspects of peripheral nerve biology. The conduction of nervous impulses along axons‚ nerve development and regeneration‚ trophic support for neurons‚ production of the nerve extracellular matrix‚ modulation of neuromuscular synaptic activity‚ and presentation of antigens. The nerve impulses
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Skinner‚ Noam Chomsky. * Describe the clinical and anatomical evidence that supports the hypothesis that different areas of the brain mediate different functions Cells of the Nervous System Reading: Gazzaniga et al. Chpt 2 Learning objectives: * Describe the 2 main types of cells in the nervous system * Describe the resting potential and how it is maintained * Describe how the action potential is generated and propagated throughout the cell * Describe the relative and
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disorder named after George Guillain (1876- 1961) and Jean Barre (1880- 1967)‚ who were the first people to describe and start to understand the disorder. GBS is a disorder in which the immune system attacks part of the peripheral nervous system‚ which is made up of nerves that extend out from the central nervous system. There are multiple names for this disorder for example it may be called‚ Acute Idiopathic Polyneuritis‚ Acute Inflammatory Demyelination‚ and Landry- Guillain-Barre syndrome. Guillain-
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amputation. The multi-factorial etiology of diabetic foot ulcers is evidenced by the numerous pathophysiologic pathways that can potentially lead to this disorder. A multicenter study attributed 63 percent of diabetic foot ulcers to the critical triad of peripheral sensory neuropathy‚ deformity‚ and trauma (Reiber‚ et al.‚ 1999). The following discusses the pathophysiology of each of the triad in limited detail. Background: Glucose is liberated from dietary carbohydrate such as starch or sucrose by hydrolysis
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The melanocortin α-melanocyte stimulating hormone (α-MSH‚ a biologically active peptide produced in POMC neurons) serves as an agonist‚ whereas AgRP is an inverse agonist at the anorectic melanocortin receptors (MC3R and MC4R) in the central nervous system. NPY elicits its physiological effects via differential activity at the G-protein coupled NPY receptors (NPY1R‚ NPY2R‚ NPY4R‚ and NPY5R) independent of melanocortin receptors [11]. Notably‚ although there are only a few thousand POMC and NPY/AgRP
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