226 World Review of Science‚ Technology and Sustainable Development‚ Vol. 4‚ Nos. 2/3‚ 2007 Nanotechnology in cancer prevention‚ detection and treatment: bright future lies ahead G. Ali Mansoori Departments of Bio and Chemical Engineering‚ University of Illinois at Chicago‚ 218 SEO‚ 851 S‚ Morgan St. (M/C 063)‚ Chicago‚ IL 60607-7052‚ USA E-mail: mansoori@uic.edu Pirooz Mohazzabi* Department of Physics‚ University of Wisconsin – Parkside‚ Kenosha‚ WI 53141‚ USA E-mail: pirooz.mohazzabi@uwp
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Are You a Good or Bad Student? Being a good student is what everyone wants to believe they are. But in reality we all know that there are bad students. I‚ myself would like to believe I am a good student but when I looked over the facts it seems that I am not a bad or a good student. The first and foremost important quality of a good student is‚ of course‚ hard working. We can’t have a good result in academic success without training and effort. The next quality is active in community. A good student
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and even repeats‚ which ultimately can result in a nonfunctional protein. The APC gene produces a protein known as the APC protein that is involved in regulating cell adhesion‚ signal transductions‚ and transcription activation. Park and Vogelstein (2003) stated that‚ “the localization of the APC protein is in the basolateral membrane of the colonic epithelial cells…” When mutations occur within this specific gene‚ it causes excessive proliferation of the colonic epithelial cells‚ which result in
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Chapter 6 Explain what free energy is and how it can be used to predict the energetic outcome of chemical reactions.Free energy is defined as the energy available to do work in any system. The free energy is denoted by the symbol G. G = H – TS* H: the energy contained in a molecule’s chemical bonds‚ called enthalpy.* TS: the energy term related to the degree of disorder in the system. T is the absolute temperature (K)‚ and S is the entropy.We can use the change in free energy to predict whether
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advanced tumor stages‚ high histological grades‚ and poor prognoses. Therefore HOTAIR may be an important target in the treatment of human osteosarcoma. It has been reported that the small nucleolar RNA host gene 12 (SNHG12) promotes cell proliferation and migration by upregulating angiomotin (AMOT) gene expression in human Osteosarcoma cells that regulates MMP-2/MMP-9 expression. Mammalian genomes encode numerous natural antisense transcripts that are at least partially complementary to their sense transcripts
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mutations are heritable 6. Definition of allele b. How process of mutation in replication leads to new alleles 7. Transgenic organism c. What it is d. How its created e. Definition of recombinant DNA 8. What gene therapy is in general Chapter 8 9. Programmed cell death =? f. Why it’s important that cell division be balanced with cell death 10. 2 main functions of apoptosis 11. Name of apoptosis-specific enzyme 12.
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Taxol is an anticancer drug produced by extraction from the bark of the pacific yew tree called Taxus brevifolia‚ which was discovered by Harvard trained botanist called Arthur Barclay in 1962 (wall and wani‚ 1995). It was renamed to paclitaxel when it was commercially developed. It is in a form of whitish crystalline powder with a melting point of 216 ºC‚ very insoluble in water‚ highly lipophilic‚ higher protein binding rate and disturbs the structure of the inner part of the cell membrane (Goldspiel
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EPIGENETICS Epigenome and Chromatin Organization E Study of heritable changes in gene activity that are not attributable to alterations in genetic sequence Potential point of crosstalk between the genome and the env’t ex: identical twins http://www.resverlogix.com/blog/wp-content/uploads/2013/02/epigenetics_large-1024x804.jpg Drivers of Epigenetic Mechanisms *Co-regulators: dictate transcription of gene http://www.nature.com/nrd/journal/v12/n12/images/nrd4154-f1.jpg Drivers of Epigenetic
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not organized Carcinoma in situ- severe dysplasia. it is still pre-cancer‚ benign tumor. Use the car analogy to describe the basic differences between oncogenes and tumor suppressor gene mutations in the development of cancer. 1. Turning on oncogens (go signals‚ step on gas‚ gas petal is stuck‚ stuck accelerator) 2. Losing tumor suppressor (cutting the brakes) 3. Becoming immortal/immortalization (endless tank of gas) 4. Loss of apoptosis- loss of cell destruction‚ cells don’t die 5. angiogenesis-
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A Project on: Negative and Positive aspects of Cigarettes ____________ Sandeep kumar NCHMCT Roll No.104686 HHA 3rd Year (2010-2013) IHM Meerut ACKNOWLEDGEMENT My project has been successful‚ thanks to my classmates. First of all I am thankful of my project guide Mr._____________ under whose guideline I was able to complete my project. I am wholeheartedly thankful to him for giving me his value able time & attention & for providing me a systematic way for completing my project in time. I would
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