High blood pressure in the lungs is called pulmonary hypertension or pulmonary arterial hypertension. Pulmonary hypertension is high blood pressure in the arteries starting from the heart towards the lungs. Pulmonary arterial hypertension (PAH) is defined as a sustained elevation of the pulmonary arterial pressure to greater than 25 mmHg at rest or greater than 30 mmHg following exercise‚ with a mean pulmonary-capillary wedge pressure (an indirect measure of left atrial pressure) of less than 15
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BLS CPR Training Guide CPR stands for Cardio Pulmonary Resuscitation. This involves the giving if life giving breaths and a series of external chest compression to person who has no breathing and no pulse. Often‚ CPR is associated with medical emergencies such as heart attack‚ drowning‚ chocking. It should be noted however that CPR is applicable to any situation where in the victim or patient has no breathing and no signs of life The Chain of Survival The chain of survival
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ARDS INTRODUCTION Acute respiratory distress syndrome (ARDS) - lung inflammation seen at the level of the alveolar capillary membrane with increased vascular permeability. ARDS results in: bilateral pulmonary edema and atelectasis despite no evidence of left heart failure (e.g.‚ normal pulmonary capillary wedge pressure (PCWP). ARDS is present when the ALI results in such severe hypoxia that at the PaO2/FIO2 ratio is 200 mm Hg or less. Approximately 10% to 15% of intensive care patients meet
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experiment‚ several physiological parameters were observed in three patients before‚ during‚ and after moderate exercise. One of these parameters was the pulmonary airflow‚ which was recorded utilizing an Airflow Transducer. This device measures airflow using slight pressure differences created by the resistance of a screen inside the device. Pulmonary airflow is the rate of movement into and out of the lungs‚ and is directly proportional to the pressure difference of the intrapulmonary pressure and
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high concentrations of oxygen‚ smoke or corrosive substances. * Localized infection (bacterial‚ fungal‚ viral pneumonia) * Metabolic disorders (pancreatitis‚ uremia) * Shock * Trauma (pulmonary contusion‚ multiple fractures‚ head injury) * Major surgery * Fat or air embolism * Systemic
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Clinic) “Acute Respiratory Distress Syndrome is diagnosed with these risk factors along with the presence of recent onset bilateral pulmonary infiltrates compatible with edema‚ critical hypoxemia‚ and the absence of evidence of a hydrostatic cause of edema‚ such as a pulmonary capillary wedge pressure (PCWP) below18 mmHg [1‚ 2]. The diagnosis therefore requires a pulmonary artery catheter‚ although a high PCWP does not exclude permeability edema. Indeed‚ ARDS is believed to result from a lung vascular
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The first type of the diseases that affect the cardiovascular system is pulmonary embolism and deep vein thrombosis which affects the veins of the leg as blood clots. Another type of CVDs is rheumatic heart disease that occurs in the heart valves and its muscle which leads to their destruction. The third type is coronary heart disease
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Antibiotics fight bacteria and the flu is cause by a virus. It can do harm if you use antibiotic to treat flu. How would left ventricle failure contribute to pulmonary edema? The pressure increases inside the left atrium and then in the veins and capillaries in our lungs. This causing fluid to be pushed through the capillary walls into the air sacs. Medical conditions that can cause the left ventricle to become
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Study Guide for the Final Exam Here are the rules: please do not call me or email me questions about the study guide. I will answer questions about the study guide during the brief review before the exam itself. You cannot memorize the answers to the questions and do well on the exam- the questions are meant to stimulate thinking‚ not to be answers. Please remember to review the chapters on shock and MODS as there are questions on this content. 1. There are several ABG questions; remember
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Case study The case study relates to Mr Brown‚ who was diagnosed with acute pulmonary oedema secondary to acute renal failure‚ due to excessive use of opioids. The assignment will address Mr Brown’s initial presentation and assessment‚ relevant past history‚ medications and the current assessment finding that had him admitted to the intensive care unit. The patient’s pathology results and drug treatment to correct his electrolyte imbalances will be discussed briefly. The assignment will discuss the
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