Tay-Sachs Disease History: The disease is named for Warren Tay (1843-1927)‚ a British ophthalmologist who in 1881 described a patient with a cherry-red spot on the retina of the eye. It is also named for Bernard Sachs (1858-1944)‚ a New York neurologist whose work several years later provided the first description of the cellular changes in Tay-Sachs disease. Sachs also recognized the familial nature of the disorder‚ and‚ by observing numerous cases‚ he noted that most babies with Tay-Sachs
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stimulus into an action potential. The eye obtains the physical stimulus in the form of light and then directs it as an electrical impulse to the brain‚ which would interpret the signal as an image. However‚ vision is a much more difficult process as the retina‚ which contains the rods or cones are located at the back of the eye‚ thus the
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Nursing Fundamentals Case Study-Sensory Directions Please read the case study. Scenario/Prompt Case Study- Sensory You are the nurse at a local assisted living center. Your client is Mrs. S.‚ an 84-year old female client. She wears eyeglasses with bifocal lenses and hearing aid in her left ear. She walks with a shuffling gait‚ using a cane for support. She wears house slippers and housedress. Mrs. S. states "My doctor says I should have my eyes looked at by an expert. It’s been a while
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Postgraduate research topics Department of Optometry and Vision Science Postgraduate research topics The following is a list of research topics offered at the doctorate (PhD)‚ masters and honours level. » Honours » Masters » PhD Honours Research Topics | Affiliation - Faculty and Dept | Supervisor/s details | Comparison of animal models of age related nuclear cataract | Optometry and Vision Science | Professor Paul Donaldson p.donaldson@auckland.ac.nz | Mapping antioxidant transporter
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Goodale‚ 1998). They used the method know as ablation or also called lesioning. Using both recordings from neurons and ablation‚ they found that properties of the ventral and dorsal streams are established by two different types of ganglion cells in the retina‚ which transmit signals to different layers of the LGN.(Goldstein‚ 2014). We know that information in the visual system progresses from the eye to the lateral
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Diabetic macular edema is a serious complication of Diabetes Milletus‚ with a prevalence of 6.81% worldwide‚ affecting 21 million people. The release of Vascular Endothelial Growth Factor‚ which is thought to be the main factor in the pathogenesis‚ leads to disruption of blood-retinal barrier‚ leading to accumulation of sub- and intra- retinal fluid‚ in the inner and outer plexiform layer. Muller cell‚ pericyte and glial cell dysfunction also contribute to the development of macular edema. Macular
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A complete assessment was performed on Bill Wilson‚ a 69 year old male. Bill appears well-groomed and oriented to person‚ place and time. With examination‚ Bill’s pupils are equal‚ alert‚ and reactive to light and no deficits with extraocular movements. Bill does wear glasses. Wisper test performed‚ no deficits found with hearing‚ no devices were worn. Bill was able to breathe out of his bilateral nostrils. His mucous membranes were pink and moist. Bill has his natural teeth with no problems observed
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The article I chose for week five is related to chapter nine notes on the eye. In the article “Lazy Eye (Amblyopia): Symptoms‚ Causes‚ Treatment” mentions certain symptoms to be aware of‚ and what causes lazy eye‚ as well as treatments available. Lazy eye is known as amblyopia it’s related to developmental problems associated within the brain. It’s usually a childhood eye problem that can affect one eye or both. If left untreated partial blindness or total blindness can occur in the eye(s) that are
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Case Study‚ Chapter 63‚ Assessment and Management of Patients With Eye and Vision Disorders 1. Ellie Lee‚ a 74-year-old patient‚ is admitted to the hospital in the same-day surgery unit for nasal surgery. The patient is legally blind. The patient was diagnosed with a genetic disease‚ recessive retinitis pigmentosa (RP)‚ which results in degeneration of the photoreceptor cells. A patient with RP has a progressive loss of visual acuity‚ progressive night blindness‚ and progressive loss of peripheral
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considerable acanthosis with broadened rete ridges causes deprivation of distant epithelial cells from blood supply and appearance becomes edematous and swollen. These necrotic cells undergo desquamation. Also leaving cleft in the surface of the epithelium. Verrucous projections are formed in this way in between clefts. At a later stage both the verrucous projections and the clefts undergo keratinization [5]. This is a case of verrucous hyperplasia with candidal superinfection in a background of
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