mammals. 3) Slide 3: Why is the nucleus of the cell in slide 3 so large? In other words‚ what is this cell doing that requires that its nucleus be so large? (Please be specific). In eukaryotic cell in slide #3 has organelles including a nucleus containing DNA and mitochondria energy organelles .Compared to the prokaryotic cell which has the DNA in the cytoplasm smaller and simpler and doesn’t contain a nucleus or other organelles‚ it does have cell membrane. Bacteria and Archea are single celled
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Cells Cells Cell division and cancer Victoria Brothwell Strayer University Cells Introduction Regular cells and cancel cells are extremely different. Depending on the cancer that one may have cancer cells have more chromosomes that are scattered which is for why cancer cells are formed. In cell division all living things obtain cells in which come from other preexisting cells. If normal cells are do not divide and make new cells then cancer will occur. In order of all cells to be
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Anemia is a disease state that is characterized by a reduction of hemoglobin (Hb) or volume of red blood cells (RBC’s). The reduction in Hb or RBC’s leads to diminished oxygen carrying ability of the blood. Many forms of anemia exist – the anemias discussed will be relevant to our patient‚ JJ‚ based on her signs/symptoms and past medical history (PMH). Anemia can be classified based on the appearance or size of the RBC. The size of the RBC can be determined by either calculating the mean corpuscular
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diagnosed with Aplastic Anemia‚ and a low chance of survival. It occurs when the bone marrow stops making a sufficient amount of healthy blood cells. As I went through the treatments I kept learning about the lab results and found out about the field of clinical laboratory science. I quickly became fascinated about the profession. The more I learned about it‚ the more I felt drawn to pursuing a career in clinical laboratory science. It took two years to get my aplastic anemia under control‚ and soon
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Equine Infectious Anemia (EIA) Equine Infectious Anemia‚ also known as swamp fever‚ is a horse disease caused by an RNA virus. This is classified in the Lentivirus genus which is characterized by a long incubation period‚ and is transmitted by bloodsucking insects (Elsevier 2013). Horses and other equidae‚ such as donkeys and zebras‚ can also contract the noncontagious‚ infectious disease. When discussing EIA‚ a clear distinction must be made between events in which the disease was contracted. Sometimes
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of the patient. Once the nurse practitioner formulates and confirms the diagnosis of pernicious anemia and potential causes what education is important for the nurse practitioner to provide or the patient? The patient should be made aware that this type of anemia is a lack of vitamin b12 uptake and lack of intrinsic factor thus making this type of anemia an autoimmune disorder. The symptoms of anemia such as: headache‚ palpitations‚ fatigue‚ pale skin and dizziness may occur as well as sensitivity
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Iron Deficiency Anemia Imagine that you lived your life to the fullest potential‚ while not having a single idea that a heart attack was right around for you. You are exhausted and have unusual cravings for ice even when you’re freezing and cannot get warm. On top of that‚ you cannot explain why you are having a shortness of breath or foggy thinking. If you imagine your life like this‚ then you are living with a condition called iron deficiency anemia. IDA is a common type of anemia‚ a condition in
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Hello Shyam‚ Thank you for your informative post about iron-deficiency anemia. Anemia is not a disease but an indication of an underlying illness‚ in which there is a decreased in the number of the red blood cells (RBCs)‚ the amount of hemoglobin (Hgb)‚ and/or the volume of RBCs (Sabol et al.‚ 2010). As we age‚ Hgb levels decrease‚ increasing the incidence of anemia (Sabol et al.‚ 2010). Iron-deficiency anemia (IDA) is the insufficiency in iron dietary intake and/or in iron absorption from different
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Introduction Pernicious anemia (PA) is a type of macrocytic normochromic anemia. It is a slow-developing disorder with the underlying cause of Vitamin B12 absorption and deficiency due to lack of intrinsic factor (IF). Vitamin B12 absorption is facilitated by IF‚ which binds B12 in the duodenum‚ forming the IF-vitamin-B12 complex‚ later recognized by receptors and absorbed in the jejunum (Anderson‚ et al.‚ 445). A consequence of vitamin B-12 deficiency is altered DNA replication and erythropoiesis
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sperm cells‚ which are also called germ cells). This type of mutation is present throughout a person’s life in virtually every cell in the body. Mutations that occur only in an egg or sperm cell‚ or those that occur just after fertilization‚ are called new (de novo) mutations. De novo mutations may explain genetic disorders in which an affected child has a mutation in every cell‚ but has no family history of the disorder. Acquired (or somatic) mutations occur in the DNA of individual cells at some
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