helpful tools. This document contains information on the PALS 2010 Guidelines. Basic Dysrhythmias knowledge is required in relation to asystole‚ ventricular fibrillation‚ tachycardias in general and bradycardias in general. You do not need to know the ins and outs of each and every one. Tachycardias need to differentiate wide complex (ventricular tachycardia) and narrow complex (supraventricular tachycardia or SVT). The course is a series of video segments then skills. The course materials
Premium Blood Blood pressure Shock
References: Cutnell‚ D. & Johnson‚ K. (2010). Introduction to Physics. John Wiley and Sons Pte Ltd. Hoboken‚ NJ. (n.a).(2010). Ventricular Fibrillation. Retrieved last September 10‚ 2010 from http://emedicine.medscape.com/article/760832-overview
Premium Series and parallel circuits Electrical resistance Resistor
evaluation. His vital signs are BP‚ 140/80; pulse‚ 148 and regular; respirations‚ 18; and SpO2‚ 97% on room air. While a colleague obtains peripheral I.V. access‚ you attach a cardiac monitor‚ which shows sinus tachycardia with frequent premature ventricular contractions (PVCs). Next‚ you use the SAMPLE mnemonic to conduct a secondary survey: Signs and symptoms: palpitations‚ shortness of breath with exertion; lightheadedness when changing from a supine to a sitting position; no chest pain‚ nausea
Premium Cardiology Myocardial infarction
Most of the cardiac injury ultimately results in development of cardiac fibrosis due to negligible regenerative capacity of heart. Cardiac fibrosis is associated with increased deposition of matrix proteins in the myocardium. The expansion of the cardiac interstitial space in absence of significant cardiomyocyte loss is “reactive interstitial fibrosis” and the formation of scar in response to myocardial infarction is called “reparative fibrosis” [10‚ 46]. Increased deposition of endomysial and perimysial
Premium Heart Myocardial infarction Blood
Etiology & Risk Factors Congestive Heart Failure can be brought on by many conditions. These conditions include Coronary Artery Disease‚ Heart Attack‚ and lastly Cardiomyopathy. Coronary artery disease can develop in humans after years over time after years of fatty deposits in the vascular system (Arteriosclerosis). After a heart attack the heart muscles are weakened and cannot perform the way they could prior to the attack. Therefore‚ as the muscles lose their pumping ability the heart cannot work
Premium Heart Hypertension Myocardial infarction
elements: 3 peaks (a‚c‚v) and 2 descents ( x‚y). ( A) wave: is reflect the atrial contraction ‚ follows the p wave on ECG waveform ‚ and it is not showed on atrial fibrillation. This wave is reflect the end diastole
Premium Heart Blood Artery
Cardiopulmonary Resuscitation (CPR) is a lifesaving technique useful in many emergencies‚ including heart attacks‚ near downing‚ when absence of breathing‚ or absence of a heart beat. In our fast paced world‚ there is a growing need for quick reaction in an emergency situation. Anyone can learn CPR. Did you know that 88% of cardiac arrest occurs at home? Knowing how to perform CPR can triple he victim’s chances of survival. If you are fearful your knowledge or abilities are not 100%‚remember
Premium Cardiac arrest Cardiopulmonary resuscitation
third-degree atrioventricular block is “a cardiac conduction system where there is no conduction through the atrioventricular node” (Budzikowski 2014). The patient will have severe bradycardia with independent atrial and ventricular rates. These patients may suffer from ventricular standstill‚ which
Premium Heart Cardiology Myocardial infarction
system of the heart. Such changes may remain asymptomatic‚ or they may progress to syncope‚ or possibly sudden death. In the event of acute myocardial infarction‚ arrhythmias are no more frequent in the elderly than in younger subjects; in fact‚ ventricular premature beats are seen less commonly
Premium Heart Blood Cardiology
Heart Diseases Arrhythmogenic right ventricular dysplasia Arrhythmogenic right ventricular dysplasia is a genetic disease in which normal heart tissue is progressively replaced with fat and scar tissue. According to St. Lukes-Roosevelt Hospital Center‚ tissue damage occurs predominantly in the right chamber of the heart. People who inherit this genetic disease may develop arrhythmias‚ or abnormal heart rhythms. Arrhythmogenic right ventricular dysplasia is a known cause of sudden death from
Premium Hypertension Heart Cardiology