"Ventricular fibrillation" Essays and Research Papers

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    Atrial fibrillation (AF) is the most common cardiac arrhythmia. Atrial fibrillation is an irregularity in the atria’s electrical impulses‚ causing a disorderly and rapid rhythm. Paroxysmal atrial fibrillation (also termed intermittent atrial fibrillation) is occurrences of AF that spontaneously halt within 7 days of starting. Episodes of AF can last from a few seconds up to a few days in Paroxysmal atrial fibrillation. The sinoatrial (SA) node is the dominant pacemaker of the heart. Located

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    Immediate post-operative ventricular bigeminy in a patient posted for ovarian cystectomy – A case report. Introduction Ventricular Bigeminy is a type of ventricular premature complex (VPC) where every normal sinus beat is followed by a ventricular ectopic. The characteristic feature here is wide QRS (0.14s) complexes with abnormal ST-T changes followed by a long pause. Ventricular arrhythmias are among the commonest arrhythmias that occurs in patient with or without heart disease.The peripheral pulse

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    (2014). Usefulness of right ventricular dysfunction to predict new-onset atrial fibrillation following coronary artery bypass grafting. The American Journal of Cardiology‚ 913-8. Retrieved from http://search.proquest.com.francis.idm.oclc.org/docview/1525978139/B9F7E9097AC14517PQ/20?accountid=4216 Summary: For many‚ atrial fibrillation may be a common postoperative complication after coronary artery bypass grafting surgery. This research study sought to identify right ventricular risk factors that may

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     Antiarrhythmic drugs primarily affect the activity of the following ion channels except  A. Cl B. Na C. K D. Ca   2. What is a life-threatening arrhythmia caused by electrical conduction disturbances?  A. Atrial fibrillation B. Ventricular fibrillation C. Ectopic foci D. Premature ventricular contraction   3. A common adverse effect from quinidine overdosage is  A. Hypertension B. Tachypnea C. Cinchonism D. Dry mouth   4. Disopyramide produces a _____ in conduction and _____ of the refractory

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    treatment. What is ventricular tachycardia? Ventricular tachycardia is a rapid heart rate originating from the ventricles (lower chambers of the heart) and produces a heart rate frequency of at least 100 beats

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    substances of acetylcholine‚ adrenaline and atropine‚ and increased frequency of stimulus. Regarding Figure 1‚ the regular heartbeat and contraction force of the toad was identified to demonstrate the difference between atrial contractions and ventricular contractions in a normal environment. In cardiac‚ skeletal‚ and some smooth muscle tissue‚ contraction occurs through a spectacle known as excitation contraction coupling (Freeman et al.‚ 2014). This process converts electrical stimulus from the

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    interval. d. QRS complex. ANS: A The P wave represents the depolarization of the atria. The P-R interval represents depolarization of the atria‚ atrioventricular (AV) node‚ bundle of His‚ bundle branches‚ and the Purkinje fibers. The QRS represents ventricular depolarization. The Q wave is the first negative deflection following the P wave and should be narrow and short. DIF: Cognitive Level: Understand (comprehension) REF: 791 TOP: Nursing Process: Assessment MSC: NCLEX: Physiological Integrity 2. The

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    about . I second is lost by this wave in traveling from the heart to the neck.2 It is probably caused by an upward movement of the auriculo-ventricular junction at the end of systole. The v wave is frequently notched and many believe that the portion preceding the notch is a stasis wave caused by the collection of blood in the auricle previous to ventricular diastole. ’ Some authors‚3 indeed‚ attribute the entire v wave to this origin. This wave is terminated by the opening of the trictuspid valves

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    are either atrial fibrillation‚ or atrial tachycardia. 48 of 49 episodes were between 150 to 190 beats per minute‚ which is within monitor only zone of the ICD. None of these episodes lasted more then one minute. He had one episode of supraventricular tachycardia‚ which was more then 190 beats per minute. This episode was terminated by antitachycardia pacing. He was asymptomatic during these tachycardias. Interrogation of the lead showed an increase in the right ventricular pacing threshold to

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    ADN 253 FINAL STUDY GUIDE 1. Describe the pathophysiology‚ assessment‚ signs and symptoms‚ nursing diagnosis‚ medical and nursing interventions for the patient with MODS. Pathophysiology • Progressive impairment of 2 or more organ systems • Caused by immune system’s uncontrolled inflammatory response to a severe illness or injury o Inflammatory response: cytokines and chemokines out of control ▪ Peripheral vasodilation = hypotension ▪ Capillary

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