For example it can be argued that the extent of DF's lesions was undermined by rejecting the possibitity that the damage might not have been completely restricted to the ventral stream (Ungerleider & Haxby, 1994). Ungerleider and Haxby argued that lesions in Brodmann's eighteenth and nineteenth area can be both dorsal and ventral. This is also extremery relevand in the case of DF due to the fact that the damage to her visual system was caused by anoxa and thus the damage would have been localised but nonetheless more diffudes than in the case with a stroke or tumor patient (Goodale ,1998). Von Karmen and Kerkhoff argued that deficits in perception of angles and axes could be related to damahe on the anterier parietal area, while deficits in estimation of position of objects would be related to posterior parietal damage. Von Karmen and Kerkhoff came up with these findings agter having tested 67 patients with parietal focal lesions. These findings imply that while it is true that damage to the dorsal stream can cause visual-spatoal and visual-motor deficits the damage cannot be fully explained by the effects. (Von Karmen & Kerkhoff,
For example it can be argued that the extent of DF's lesions was undermined by rejecting the possibitity that the damage might not have been completely restricted to the ventral stream (Ungerleider & Haxby, 1994). Ungerleider and Haxby argued that lesions in Brodmann's eighteenth and nineteenth area can be both dorsal and ventral. This is also extremery relevand in the case of DF due to the fact that the damage to her visual system was caused by anoxa and thus the damage would have been localised but nonetheless more diffudes than in the case with a stroke or tumor patient (Goodale ,1998). Von Karmen and Kerkhoff argued that deficits in perception of angles and axes could be related to damahe on the anterier parietal area, while deficits in estimation of position of objects would be related to posterior parietal damage. Von Karmen and Kerkhoff came up with these findings agter having tested 67 patients with parietal focal lesions. These findings imply that while it is true that damage to the dorsal stream can cause visual-spatoal and visual-motor deficits the damage cannot be fully explained by the effects. (Von Karmen & Kerkhoff,