If the patient was known to have a partial obstruction on the right carotid artery 2 years ago, we can assume a thrombus which originated from this obstruction was later lodged downstream causing the stroke in the right hemisphere. It is also evident that the partial obstruction in the internal carotid became further constricted overtime because of the buildup of plaque and atherosclerosis further …show more content…
The patient’s visual field defects are characterized as left homonymous hemianopia with macular sparing which is defined as visual field loss in the left ipsilateral region of oculus unitas. The nerve bundles which are extended from the retina consist of the ganglion cell axons which make up the optic tract. The semidecassation is the incomplete crossing of nerve fibers at the optic chiasm which results in each optic tract containing half of the ipsilateral optic nerve and one half of the contralateral optic nerve such that the ipsilateral hemifield from each eye correlates to the contralateral hemisphere (Leff 2004). Due to this phenomenon we can infer that the left homonymous hemianopia is due to a lesion in the right optic tract or the right occipital lobe as shown below in Figure