Nile Tilapia Fish Lab Report
Results
Mortality rates, clinical signs and postmortem findings
Mortalities in all groups were recorded throughout the experiment and summarized in Table 1. No mortalities were recorded in the control group throughout the experimental period. Table 1 show that the highest mortality rate occurred in Nile tilapia fish in the S. iniae+ C. albicans infected group (76.19%), followed by those infected with S. iniae alone (61.9%) and then the C. albicans infected group (57.14%). Clinically, the S. iniae-infected fish exhibited loss of appetite, loss of equilibrium with erratic movement, swam near to the surface of water, detachment of scales, hemorrhages on the different parts of body surface, slight abdominal dropsy, slight exophthalmia (unilateral …show more content…
As well there was white pulp depletion in all in infected groups, particularly in the S. iniae+C. albicans-infected group (Fig. 5).
Brain
Brain of fish in the S. iniae-infected group showed congestion of the blood vessels and small shrunken pyknotic neurons during the 1st week of the experiment (Fig. 6a). At the end of the experiment, there were pyknosis and depletion in the number of purkinje cells besides pericellular edema. Brain of the C. albicans-infected fish had congested blood vessels (Fig. 6b) with mild neuronal pyknosis and the S. iniae+C. albicans-infected fish exhibited similar lesions to those of the S. iniae-infected group (Fig. 6c).
Intestine
Intestine of fish in the S. iniae-infected group showed submucosal eosinophilic granular cells (EGCs) infiltration during the 1st week of the experiment (Fig. 7a). There was hyperplasia of the goblet cells in the C. albicans-infected group during the 2nd week of the experiment (Fig. 7b). Regarding to the S. iniae+C. albicans-infected fish, there were focal necrosis of the intestinal epithelium with mononuclear inflammatory cell infiltrations (Fig. 7c), particularly during the 2nd week of the
Mortality rates, clinical signs and postmortem findings
Mortalities in all groups were recorded throughout the experiment and summarized in Table 1. No mortalities were recorded in the control group throughout the experimental period. Table 1 show that the highest mortality rate occurred in Nile tilapia fish in the S. iniae+ C. albicans infected group (76.19%), followed by those infected with S. iniae alone (61.9%) and then the C. albicans infected group (57.14%). Clinically, the S. iniae-infected fish exhibited loss of appetite, loss of equilibrium with erratic movement, swam near to the surface of water, detachment of scales, hemorrhages on the different parts of body surface, slight abdominal dropsy, slight exophthalmia (unilateral …show more content…
As well there was white pulp depletion in all in infected groups, particularly in the S. iniae+C. albicans-infected group (Fig. 5).
Brain
Brain of fish in the S. iniae-infected group showed congestion of the blood vessels and small shrunken pyknotic neurons during the 1st week of the experiment (Fig. 6a). At the end of the experiment, there were pyknosis and depletion in the number of purkinje cells besides pericellular edema. Brain of the C. albicans-infected fish had congested blood vessels (Fig. 6b) with mild neuronal pyknosis and the S. iniae+C. albicans-infected fish exhibited similar lesions to those of the S. iniae-infected group (Fig. 6c).
Intestine
Intestine of fish in the S. iniae-infected group showed submucosal eosinophilic granular cells (EGCs) infiltration during the 1st week of the experiment (Fig. 7a). There was hyperplasia of the goblet cells in the C. albicans-infected group during the 2nd week of the experiment (Fig. 7b). Regarding to the S. iniae+C. albicans-infected fish, there were focal necrosis of the intestinal epithelium with mononuclear inflammatory cell infiltrations (Fig. 7c), particularly during the 2nd week of the