Badescu Skin Care (2012). Photo-aging. Retrieved from http://www.mariobadescu.com/photoaging. Robinson‚ JK. Sun exposure‚ sun protection‚ and vitamin D. JAMA 2005; 294:1541-43. Rogers‚ HW‚ Weinstock‚ MA‚ Harris‚ AR‚ et al. Incidence estimate of non-melanoma skin cancer in the United States‚ 2006. Arch Dermatol 2010; 146(3):283-287. Skin Cancer Foundation (2013). What is Photo-aging? Retrieved from http://www.skincancer.org/healthy-lifestyle/anti-aging/what-is-photoaging. Zhang M‚ Qureshi AA‚ Geller
Premium Ultraviolet Sun tanning Sunscreen
Arguments on Tanning There are many different opinions on whether or not tanning is good for you. Some believe that tanning may cause skin cancer‚ leading to many deaths. On the other hand‚ many people that tan see tanning as a way to receive the vitamin D that one needs in their day-to-day lives. There are also various reasons as to why one tans. One major reason is to darken their skin and by doing this‚ they gain confidence. Others truly go tanning to soak up the vitamin D that they need for
Premium Ultraviolet Sun tanning Sunscreen
Cancer Epidemiology Cancer is not a single disease; it is a myriad or collection of diseases with many different manifestations as there are many different types of tissues and cell types Cancer is a disease of abnormal gene expression. Cells are constantly evolving and have the natural selection pressures to change Common biological properties of tumor cells: 1) Deregulated clonal cell growth- all cells in a tumor originated from one single cell that acquired mutations that made it deregulated
Premium Cancer DNA
reductions; they were checked for any atypical material‚ although because the reductions were for cosmetic material‚ atypical material was not expected. I also observed the dissection of fatty tissue in a patient with duct carcinoma. It was pretty tough watching the carcinoma being dissected; whilst it was interesting to watch from an educational perspective‚ it was also upsetting
Premium
1. Judy has fair skin / red hair‚ her skin has less melanin than Mariah’s. This puts her at higher risk for melanoma than Mariah. 2. Judy observed that her mole was larger and more jagged than she remembered. It was discolored‚ with a raised purplish-black dot. This occurred because cells called melanocytes produce more pigment. Discolored or multicolored moles are considered atypical. 1. Benign tumors have no potential to spread‚ and will not affect the organ’s function. However removal of
Premium Cancer Ultraviolet
Integumentary System Study Guide 1. Which portion of the epidermis has actively reproducing cells? Stratum Basale 2. Are keratin levels higher in superficial or deep epidermis? Superficial 3. Blood vessels and nerves are present in which layer of skin? Dermis 4. In what layer of skin can you find melanocytes? Epidermis (stratum basale) 5. What structure pulls the hair follicle into an upright position? Arrector Pili 6. What structure is responsible for whorled ridges on epidermal surfaces? Dermal
Premium Skin
DIAGNOSIS Basal cell carcinoma; of the right ear. POSTOPERATIVE DIAGNOSES Basal cell carcinoma; of the right ear. PROCEDURE PERFORMED Excision of basal cell carcinoma on the right ear; measuring 3.5 by 2 centimeters in length and width. A frozen section‚ as well as a full thickness of skin graft taken from the right supraclavicular region; for coverage measure 3.5 by 2 centimeters. ANESTHESIA MAC anesthesia INDICATIONS 79 year old male‚ who has a history of basal cell carcinoma‚ on the right posterior
Premium Surgery Basal cell carcinoma
of the breast. Such as‚ the mammary gland‚ lobules‚ areola‚ nipple‚ lactiferous ducts‚ and the subcutaneous fat. It is known‚ that there are two types of breast cancer: Ductal Carcinoma- starts in the tubes (ducts) that move milk from the breast to the nipple.1 This is the most common cancer in woman. Lobular Carcinoma- starts in the parts of the breast‚ called lobules‚ which produce milk.2 Depending of the severity of the breast cancer‚ it falls into two grading or categories. Those categories
Premium Breast cancer Cancer Breast
Benign Prostatic Hyperplasia Benign prostatic hyperplasia (BPH)‚ also known as benign prostatic hypertrophy‚ is a histologic diagnosis characterized by proliferation of the cellular elements of the prostate. Cellular accumulation and gland enlargement may result from epithelial and stromal proliferation‚ impaired preprogrammed cell death (apoptosis)‚ or both. BPH involves the stromal and epithelial elements of the prostate arising in the periurethral and transition zones of the gland. The hyperplasia
Premium Stroke Cancer
receptor). Increasing evidence is showing that the greater number of variations in this gene‚ the greater the risk for melanoma. It also carries a more moderate risk that the CDKN2A mutation which is the most common inherited mutation‚ usually these people have a large number of irregularly shaped moles. Approximately 70 percent of people who have CDKNA gene mutation will develop melanoma. What genes control the color of skin‚ hair‚ and eyes? The MC1R (Melanocortin 1 receptor) gene is partly responsible
Premium Mutation Ultraviolet DNA