What Effect Would Cyanide Have On The Electron Transport Chain
What effect would cyanide have on the electron transport chain and the production of atp? Explain your answer.
When the oxygen are absence, the ETC will stops working and ultimately the whole reaction to create ATP will stop. Cyanide will disrupts oxygen from binding to the final molecule in the electron transport chain. The electron from NADH cannot be transferred to NAD+ due to the failure for bringing electron transported to the oxygen. To make it simple Cyanide stops NADH from being used to make NAD+. Hence, the mitochondria cannot make ATP.
Given what you now know about the action of cyanide on cellular respiration, explain why the patients died of lack of oxygen while their blood oxygen levels were normal?
Oxygen are significant for ETC so that the reaction will continue to function. ETC will halts once the body are lacks with oxygen. The ATP no longer be formed, and death occurs frequently. In these patients, cyanide blocked the ETC so that electron cannot be remove eventhough the body have oxygen. so even though it was present in normal levels in the blood cells, oxygen could not enter the mitochondria due the cyanide preventing it and could not perform its function, the patients were unable to utilize it and thus died from oxygen's lack of function. …show more content…
Why or why not?
No it would not. It would not help the mitochondria as the cyanide still would not allow the oxygen to enter into it. As we know, Cyanide poisoning affects the ability of cells to use oxygen for aerobic respiration. The tissues will not be able to use it no matter how much oxygen they have in their blood. So, giving a person poisoned with cyanide extra oxgen or artificial respiration are useless because oxygen is already excess.
Looking back at the information you have about the people before they got sick, can you suggest a possible source of the cyanide poisoning? How should public health officials and police respond to this
When the oxygen are absence, the ETC will stops working and ultimately the whole reaction to create ATP will stop. Cyanide will disrupts oxygen from binding to the final molecule in the electron transport chain. The electron from NADH cannot be transferred to NAD+ due to the failure for bringing electron transported to the oxygen. To make it simple Cyanide stops NADH from being used to make NAD+. Hence, the mitochondria cannot make ATP.
Given what you now know about the action of cyanide on cellular respiration, explain why the patients died of lack of oxygen while their blood oxygen levels were normal?
Oxygen are significant for ETC so that the reaction will continue to function. ETC will halts once the body are lacks with oxygen. The ATP no longer be formed, and death occurs frequently. In these patients, cyanide blocked the ETC so that electron cannot be remove eventhough the body have oxygen. so even though it was present in normal levels in the blood cells, oxygen could not enter the mitochondria due the cyanide preventing it and could not perform its function, the patients were unable to utilize it and thus died from oxygen's lack of function. …show more content…
Why or why not?
No it would not. It would not help the mitochondria as the cyanide still would not allow the oxygen to enter into it. As we know, Cyanide poisoning affects the ability of cells to use oxygen for aerobic respiration. The tissues will not be able to use it no matter how much oxygen they have in their blood. So, giving a person poisoned with cyanide extra oxgen or artificial respiration are useless because oxygen is already excess.
Looking back at the information you have about the people before they got sick, can you suggest a possible source of the cyanide poisoning? How should public health officials and police respond to this