echocardiogram and possible cardiac bypass. Five days previously‚ Cecil had presented to an emergency department with complaints of shortness of breath on exertion‚ pneunoperiteum (PNO) and orthopnea. After being treated at the previous hospital for acute pulmonary odema (APO)‚ secondary to a Non-Stemi with a Troponin level of 0.88l/min and was treated accordingly with Frusemide and continuous positive airway pressure (CPAP). He has no previous history of cardiac arrhythmias but he does have a history
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Give an account of how a poor diet and obesity can increase the chance of a person developing Coronary Heart Disease (CHD) A good diet consists of consuming a full range of all the various nutrients needed for proper function of the body and bodily systems. When someone consumes the correct nutrients in the correct proportions‚ they are considered to have a “Balanced Diet”. However‚ when someone is not consuming the correct nutrients or not consuming the correct amount of nutrients they are
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neurons 8. Myocardial contraction plays an important role in phase _____‚ where an influx of _____ ions predominate. A. 1‚ Na B. 1‚ Ca C. 2‚ Na D. 2‚ Ca 9. A prolongation of the QT interval can lead to all of the following except A. Hypoglycemia B. Hypotension C. Fainting D. Ventricular arrhythmias 10. Beta blockers are mainly indicated for _____ and for prevention of recurrent _____. A. Supraventricular arrhythmias‚ ischemia B. Supraventricular arrhythmias‚ myocardial infarctions
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continues to be a major public health issue worldwide. This paper is a case study report about C.B‚ a 60 year old woman of East Indian descent. She has a medical history of hypertension‚ ischemic heart disease‚ asthma and myocardial infarction ×4 and is currently diagnosed with “acute exacerbation of congestive cardiac failure”. According to (Woods‚ et. Al‚ 2010)‚ heart failure the pathphysiologic state in which an abnormality of cardiac function is responsible for inadequate systemic function. Further
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coronary arteries that define a heart attack (myocardial infarction) or other common non-infectious causes. Myocarditis may or may not include death (necrosis) of heart tissue. When the heart is involved in an inflammatory process‚ often caused by an infectious agent‚ myocarditis is said to be present Inflammation may involve the myocytes‚ interstitium‚ vascular elements &/or pericardium Characterized by isolated pockets of inflamed & necrotic myocardial cells Usually of sudden onset CAUSES
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EXERCISE 29 t-TEST FOR INDEPENDENT GROUPS I STATISTICAL TECHNIQUE IN REVIEW The t-test is a parametric analysis technique used to determine significant differences between the scores obtained from two groups. The t-test uses the standard deviation to estimate the standard error of the sampling distribution and examines the differences between the means of the two groups. Since the t-test is considered fairly easy to calculate‚ researchers often use it in determining differences between two groups
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alb—albumin alt dieb—alternate days (every other day) am—morning AMA—against medical advice amal—amalgam amb—ambulate‚ walk AMI—acute myocardial infarction amt—amount ANS—automatic nervous system ant—anterior AOx3—alert and oriented to person‚ time‚ and place Ap—apical AP—apical pulse approx—approximately aq—aqueous ARDS—acute respiratory distress syndrome AS—left ear ASA—aspirin asap (ASAP)—as soon as possible as tol—as tolerated ATD—admission‚ transfer‚
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Ischemic Heart Disease (IHD) Coronary heart disease (CHD)‚ also known as ’ischemic heart disease’‚ is the most common form of heart disease. There are two major clinical forms – heart attack (often known as ’acute myocardial infarction’ or AMI) and angina. Coronary heart disease is the term that describes what happens when your heart’s blood supply is blocked or interrupted by a build-up of fatty substances in the coronary arteries. Over time‚ the walls of your arteries can become clogged
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NS 3205 Study Guide for Final Exam Chapter 42 Care of Patients with Hematologic Problems 1. Identify the etiologies and clinical manifestations common to all types of anemia. (See Table 42-1 p 870 and Chart 42-1 p 871) Common Cause Sickle cell disease: autosomal recessive inheritance of two defective gene alleles for hemoglobin synthesis Glucose-6-phosphate dehydrogenase (G6PD) deficiency anemia: X-linked recessive deficiency of enzyme G6PD Autoimmune hemolytic anemia: abnormal
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render care for the patient‚ the student will be able to: * Discuss the overview of the disease in the review of the Related Literature. * Identify its clinical manifestation. * Present the Anatomy and Physiology * Trace the pathophysiology of the disease * Establish a good and therapeutic nurse-patient interaction. * Monitor patient’s progress. * Determine the status of the patient through: a. General Data b. Physical
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