Surface Tension Lab Report
Shakespeare Sance
Homework # 4
The factors that would cause the lung to collapse are elastic recoil and high surface tension. And the factors that prevent the lungs from collapse are negative intrapulmonary pressure and surfactant.
During expiration : volume increase and pressure decrease
During inspiration : volume decrease and pressure increase
The role of surfactant is to decrease surface tension by more than 40 times.
And it's effect on surface tension is to prevent them from PO2 in alveolar : 104
PO2 in venous blood : 40
PO2 in atmosphere : 160
PO2 in arterial : 100
PO2 in expired : 120
PO2 in inspired : 160
Co2 is 20 times more soluble in water than oxygen
Anatomical dead space : the section where there is no gas …show more content…
exchange
Physiological dead space : the sum of anatomical dead space and alveolar Dead space
And it would only increase if a person is sick for example suffering from asthma .
Compliance in respiratory care: how much stretch you have how much changes in pressure naturally the lungs have a high compliance .
Low compliance : pulmonary edema and cystic fibrosis
High compliance : emphysema : where a person can get air in and can't get it out
O2-Hb dissociation curve:when the curve mov to the right there is less saturation of hemoglobin . When is shift to the left less oxygen release more oxygen bound to hemoglobin.
Situations that would cause more o2 to release are high temperature, low PH , exercise , high co2.
Way O2 are transport: hemoglobin and plasma
Way CO2 are transport : plasma , bicarbonate ion , and carbamino-compounds
Chloride shift : exchange of bicarbonate for chloride across the red blood cell
The role of the phrenic nerve : excite the diaphragm and intercostal muscles , causing breathing movements.
Anatomy respiratory membrane: a simple layer of squamous epithelial of alveolus and a simple layer of squamous epithelial of the capillary very thin, together the alveolar and capillary walls and their fused basement membranes form the respiratory membrane.
Increase in thickness of the respiratory membrane would decrease gas exchange or imputed gas …show more content…
exchange.
Inspiratory center: comparable to the SA node this center sets the rhythmicity of respiration by spontaneously depolarizing and stimulating the phrenic nerve to cause inspiration.
Expiratory center: it's caused as the inspiration center no longer stimulate the diaphragm stop expiratory allows us to respirate.
Pneumotaxic center: this center inhibits the inspiratory center to limit the period of inspiration.Damage to the pneumotaxic center may cause prolonged inspirations.
Apneustic center: this center stimulates the inspiratory center and is usually inhibited by the pneumotaxic center.
Cortical nephron: has short loop of henle and glomerelus further from the corticomedullary junction.efferent arteriole supplies peritubular capillaries
Justamedullary nephron: has long loop of henle and glomerulus closer to the corticomedullary junction.efferent arteriole supplies vasa recta.
The filtration membrane: basement membrane, podocytes, and glomerular endothelium
Pressure forces fluid out the glomerulus: glomerular blood hydrostatic pressure
Force oppose fluid to move out : blood colloid osmotic pressure Afferent vasodilate: blood flow increase , blood pressure increase and GFR increase
Efferent vasodilate: blood flow decrease, blood pressure decrease and GFR decrease
Afferent vasoconstriction: blood flow decrease, blood pressure decrease and GFR decrease.
Efferent vasoconstriction: blood flow increase , blood pressure increase and GFR increase.
The role of renin is to cut angiotensinogen to angiotensin 1 and it's secreted when there is sense of low blood pressure.
Everything that are reabsorbed back into the blood has tubular maximum and you exceed the T max , the exceed remind in the urine.
The major hormones that regulates potassium secretion is aldosterone
Acidosis: PH under 7.35
Alkalosis: PH over 7.45
PH: measures free hydrogen ions range from 0 to 14 there is an inverse relationship between hydrogen ions and PH as hydrogen ion goes up PH went down
Three majors ways to buffeting urine : chemical buffer system, bicarbonate buffer system and protein buffer system.
Hypoventilation : PH went down
Hyperventilation : PH goes up
ECF: fluid inside the cell
ICF : fluid outside the cell
More sodium ECF than ICF
More potassium ICF than ECF
Countercurrent system: fluid flowing in parallel tubes in opposite
direction
And you will find one at the
They shouldn't no red blood , protein , bile pigment and no glucose
Intense SNS no urine
Little SNS blood pressure goes up GFR goes up
PSNS : more urine
Sensible : sweat you can actually see they are hypotonic and has electrolytes
Insensible : sweat you don't see come from your skin and you ears .
Homework # 4
The factors that would cause the lung to collapse are elastic recoil and high surface tension. And the factors that prevent the lungs from collapse are negative intrapulmonary pressure and surfactant.
During expiration : volume increase and pressure decrease
During inspiration : volume decrease and pressure increase
The role of surfactant is to decrease surface tension by more than 40 times.
And it's effect on surface tension is to prevent them from PO2 in alveolar : 104
PO2 in venous blood : 40
PO2 in atmosphere : 160
PO2 in arterial : 100
PO2 in expired : 120
PO2 in inspired : 160
Co2 is 20 times more soluble in water than oxygen
Anatomical dead space : the section where there is no gas …show more content…
exchange
Physiological dead space : the sum of anatomical dead space and alveolar Dead space
And it would only increase if a person is sick for example suffering from asthma .
Compliance in respiratory care: how much stretch you have how much changes in pressure naturally the lungs have a high compliance .
Low compliance : pulmonary edema and cystic fibrosis
High compliance : emphysema : where a person can get air in and can't get it out
O2-Hb dissociation curve:when the curve mov to the right there is less saturation of hemoglobin . When is shift to the left less oxygen release more oxygen bound to hemoglobin.
Situations that would cause more o2 to release are high temperature, low PH , exercise , high co2.
Way O2 are transport: hemoglobin and plasma
Way CO2 are transport : plasma , bicarbonate ion , and carbamino-compounds
Chloride shift : exchange of bicarbonate for chloride across the red blood cell
The role of the phrenic nerve : excite the diaphragm and intercostal muscles , causing breathing movements.
Anatomy respiratory membrane: a simple layer of squamous epithelial of alveolus and a simple layer of squamous epithelial of the capillary very thin, together the alveolar and capillary walls and their fused basement membranes form the respiratory membrane.
Increase in thickness of the respiratory membrane would decrease gas exchange or imputed gas …show more content…
exchange.
Inspiratory center: comparable to the SA node this center sets the rhythmicity of respiration by spontaneously depolarizing and stimulating the phrenic nerve to cause inspiration.
Expiratory center: it's caused as the inspiration center no longer stimulate the diaphragm stop expiratory allows us to respirate.
Pneumotaxic center: this center inhibits the inspiratory center to limit the period of inspiration.Damage to the pneumotaxic center may cause prolonged inspirations.
Apneustic center: this center stimulates the inspiratory center and is usually inhibited by the pneumotaxic center.
Cortical nephron: has short loop of henle and glomerelus further from the corticomedullary junction.efferent arteriole supplies peritubular capillaries
Justamedullary nephron: has long loop of henle and glomerulus closer to the corticomedullary junction.efferent arteriole supplies vasa recta.
The filtration membrane: basement membrane, podocytes, and glomerular endothelium
Pressure forces fluid out the glomerulus: glomerular blood hydrostatic pressure
Force oppose fluid to move out : blood colloid osmotic pressure Afferent vasodilate: blood flow increase , blood pressure increase and GFR increase
Efferent vasodilate: blood flow decrease, blood pressure decrease and GFR decrease
Afferent vasoconstriction: blood flow decrease, blood pressure decrease and GFR decrease.
Efferent vasoconstriction: blood flow increase , blood pressure increase and GFR increase.
The role of renin is to cut angiotensinogen to angiotensin 1 and it's secreted when there is sense of low blood pressure.
Everything that are reabsorbed back into the blood has tubular maximum and you exceed the T max , the exceed remind in the urine.
The major hormones that regulates potassium secretion is aldosterone
Acidosis: PH under 7.35
Alkalosis: PH over 7.45
PH: measures free hydrogen ions range from 0 to 14 there is an inverse relationship between hydrogen ions and PH as hydrogen ion goes up PH went down
Three majors ways to buffeting urine : chemical buffer system, bicarbonate buffer system and protein buffer system.
Hypoventilation : PH went down
Hyperventilation : PH goes up
ECF: fluid inside the cell
ICF : fluid outside the cell
More sodium ECF than ICF
More potassium ICF than ECF
Countercurrent system: fluid flowing in parallel tubes in opposite
direction
And you will find one at the
They shouldn't no red blood , protein , bile pigment and no glucose
Intense SNS no urine
Little SNS blood pressure goes up GFR goes up
PSNS : more urine
Sensible : sweat you can actually see they are hypotonic and has electrolytes
Insensible : sweat you don't see come from your skin and you ears .