Community Acquired Pneumonia Dora E. Wiskirchen‚ PharmD‚ BCPS PHCY 836 – Day 5 PM Learning Objectives Identify risk factors for the development of pneumonia. Explain the pathophysiology‚ signs and symptoms‚ most common bacterial etiologies and associated resistance patterns‚ severity of illness scoring systems‚ and diagnostic techniques for pneumonia. Define atypical pneumonia and characterize patients who may be at risk for developing this type of pneumonia. Evaluate pharmacologic
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means the antibodies are produced and complement proteinsC1‚2 and 4 are activated and in turn activated C3. The alternative pathway is when C3 and factors B‚D‚ and P interact on the surface of the pathogen and C3 is again turned on and at this point it is split into C3a and C3b. When C3b binds to the pathogen the inflammation we all know and love becomes present. Fever is a response by the neurons in the hypothalamus in response to pyrogens which are secreted by macrophages and leukocytes. Pain is caused
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Activity 1.1.3 Using DNA to Identify Pathogens Describe the limitations of traditional methods of identifying bacteria. Traditional methods include judging by phenotypic characteristics. This could be a problem if two bacteria looked similar because they could easily be confused. Summarize the goal of each of the six parts of the lab. Sample Prep First we need to extract the DNA from the bacterial wall PCR Amplification Create millions of copies of the initial DNA PCR Purification
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causing s leaf spot (GLS). According to Ward and Nowell (1997) in the year 1991 in South Africa it was recognised as one of the imperatives pathogen of economic taken into consideration in the maize industry. Considering its capability the
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against disease. These prevent disease from entering the mammal’s body by acting as a physical barrier. Cilia which line the airways‚ as well as sweeping cells in the lungs and urinogenital tract‚ play a role in the non-specific defence. Cilia push pathogen-containing mucous out of the body. The acid of the stomach is an example
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Urinary tract infections refer to the presence of pathogenic microorganisms within the urinary tract and its classification is usually done on the basis of infection sites:-bladder [cystitis]‚ kidney [pyelonephritis]‚ or urine [bacteriuria] and also can be asymptomatic or symptomatic (Quigley R 2009‚ Schlager TA2001). They are among the most commonly acquired bacterial infections and they account for an estimated 25-40% of the nosocomial infections (Bagshaw SM‚ Laupland KB 2006). Urinary tract infection
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individual has been infected with a pathogen? More white blood cells are being made to fight off the pathogen. 7. In regions where malaria is endemic‚ some people build up immune resistance to the malaria pathogen. Which WBCs are responsible for the immune response against pathogens? How do they functions? White blood cells or leukocytes are vital cells of the immune system protecting the human body against infections‚ bacteria‚ microbes‚ viruses and pathogens. These cells are produced in the stem
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insignificant. However‚ plants have adapted and evolved chemical‚ physical‚ and protein based mechanisms to protect themselves against invaders. A plant’s first line of defense against pathogens is their innate immunity. The “innate-immune system” is a non-specific immune system and responds to any recognizable pathogens in a general manner. An example of an innate immune defense would be a plant’s outer layer of waxy skin or cuticle that protects it from bacteria‚ the same way that a human’s epithelial
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from plasma/interstitial fluid. Does not have plasma proteins What does the Lymphatic System do? Returns interstitial fluid and leaked plasma proteins to the blood stream It produces‚ maintains‚ and distributes lymphocytes Protects against pathogens Immune system is all body cells and tissues involved in immunity (includes integumentary‚ digestive‚ cardio‚ respiratory‚ etc) Circulation: lymphatic capLymph collecting vessles (w/valves)Lymph nodeLymph trunkLymph duct heart Lymph vessels
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Our main goal was to test organisms’ resistance and susceptibility to bacteria‚ and whether the cell wall of bacteria exposed the membrane to pathogen or protect it from pathogens. I thought P. aeruginosa was gram-positive and therefore it would be susceptible. That was my hypothesis.We divided antiseptic agar plate into quadrants and divided antibiotics agar plate into six areas. We wrote the organisms’ name‚ date‚ and the temperature on the bottom of the agar plates. For antiseptic‚ our group used
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