INTRODUCTION Definition; It is a complex clinical syndrome that can result from any cardiac disorder that impairs the ability of the ventricle to deliver adequate quantities of blood to the metabolizing tissues during normal activity or at rest. Causes 1. Although the disease occurs most commonly among the elderly (80% of patients hospitalized with CHF are > 65 years of age)‚ it may appear at any age as a consequence of underlying cardiovascular disease. 2. There currently is no single
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Nursing Diagnosis # 1 Ineffective breathing pattern related to decreased oxygen saturation‚ poor tissue perfusion‚ obesity‚ decreased air entry to bases of both lungs‚ gout and arthritic pain‚ decreased cardiac output‚ disease process of COPD‚ and stress as evidenced by shortness of breath‚ BMI > 30 abnormal breathing patterns (rapid‚ shallow breathing)‚ abnormal skin colour (slightly purplish)‚ excessive diaphoresis‚ nasal flaring and use of accessory muscles‚ statement of joint pain‚ oxygen
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The aim of this assignment is to critically discuss the nursing assessment individualised care and nursing interventions of the acutely ill patient. The patient discussed developed severe sepsis due to a urinary tract infection and her condition deteriorated during the recovery process in the nurse’s care. Lovick (2009) defines sepsis ‘as a known or suspected infection accompanied by evidence of two or more of the SIRS criteria’. SIRS is outlined as a ‘systemic inflammatory response’ consisting of
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symptoms of acute coronary syndrome and a history that is suggestive for PE. The patient experiencing PE will generally present with difficulty breathing‚ chest pain on inspiration‚ and palpitations. Patient assessment will typically show signs of decreased oxygen saturation (SpO2)‚ cyanosis‚ rapid breathing‚ and a rapid heart rate. Severe cases of PE may include syncope‚ low blood pressure‚ and sudden death (Goldhaber‚ Pulmonary Thromboembolism). In the instance of a significant occlusion of the pulmonary
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Sodium‚ Hypertension‚ and an Explanation of "Lag Phenomenon" in Hemodialysis Patients Author: Zbylut Twardowski‚ MD‚ PhD I. INTRODUCTION Healthy kidneys clean your blood by removing excess fluid‚ minerals‚ and wastes. They also make hormones that keep your bones strong and your blood healthy. When your kidneys fail‚ harmful wastes build up in your body‚ your blood pressure may rise‚ and your body may retain excess fluid and not make enough red blood cells. When this happens‚ you need treatment
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45 degrees. The nurse knows this finding indicates a. decreased fluid volume. b. jugular vein atherosclerosis. c. increased right atrial pressure. d. incompetent jugular vein valves. ANS: C The jugular veins empty into the superior vena cava and then into the right atrium‚ so JVD with the patient sitting at a 45-degree angle reflects increased right atrial pressure. JVD is an indicator of excessive fluid volume (increased preload)‚ not decreased fluid volume. JVD is not caused by incompetent jugular
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The patient’s fluid and diet intake had decreased‚ he slept more frequently‚ and was found to be confused at times; Mr Brown had high tolerance to opioids which later progressed to opioid toxicity. As noted age effects all elements of the body’s system and increased age did have an impact on Mr
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Understanding Health Related Physical Fitness 1. What are 8 different health reasons to exercise given in the reading? ■increases longevity ■reduces risk for heart disease ■reduces risk for diabetes ■maintains work capacity ■helps reduce high blood pressure
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1. Citation: Authors: Stefan Miedler‚ Andreas Fahlman‚ Monica Valls Torres‚ Teresa Alvaro Alvarez‚ and Daniel Garcia Parraga Year published: 2015 Title of the Article: Evaluating cardiac physiology through echocardiography in bottlenose dolphins: using stroke volume and cardiac output to estimate systolic left ventricular function during rest and following exercise Journal: Journal of Experimental Biology Volume: 218 Page numbers: 3604-3610 2. Research Sponsor: a. The authors of this experiment
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stress hormones‚ such as adrenaline and noradrenaline. These hormones stimulate the heart‚ leading to an increase in heart rate(Chu et al.‚ 2024). Tachycardia serves as a compensatory mechanism to maintain adequate tissue perfusion by increasing cardiac output. Additionally‚ hypertension is commonly observed in patients with TBI. The sympathetic nervous system is activated in response to the injury‚ leading to peripheral vasoconstriction and increased systemic vascular resistance (Jang et al.‚ 2020)
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