& Gersh‚ B. (2009). Chronic coronary artery disease: diagnosis and management. Mayo Clinic Proceedings‚ 84(12)‚ 1130-1146. doi:10.4065/mcp.2009.0391 DiSabatino‚ A. J & Butcher‚ L. (2008). Nusing management: coronary artery disease and acute coronary syndrome. In Brown‚ D.‚ Edwards‚ H.‚ Lewis‚ S.‚ Heitkemper‚ M.‚ Dirkson‚ S.‚ O’Brien‚ P.‚ & Bucher‚ L. (Eds.). Lewis’s medical-surgical nursing: assessment and management of clinical problems. (2nd ed.). Sydney: Mosby. Faxon‚ D. P.
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pressure and tension are greater on the left side of the heart. When systolic dysfunction affects the right side of the heart‚ blood flow through the pulmonary circulation is reduced. The most common precipitating cause of systolic dysfunction is acute myocardial
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elevated cholesterol‚ and poorly controlled type II diabetes. He has recently quit smoking and drinks up to 12 beers on the weekend. He lives a sedentary lifestyle and works long hours in Information Technology. His father died at 65 from an Acute Myocardial Infarction (AMI)‚ and his mother lives next door and requires assistance with Activities of Daily Living (ADL’s).
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Focus Questions #1 1. Relate each client’s current manifestations to the pathophysiology of shock to determine what type of shock the client could be experiencing. Shock is a syndrome characterized by decreased tissue perfusion and impaired cellular metabolism. The patient Richard Tanner has been admitted to the CCU for r/o myocardial infarction. The patient has not prior history of cardiac problems though he has been treated for the last 5 years for cholesterol totaling 285 (HDL 35‚ LDL 212)
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arms will tell us that it might be acute pericarditis. In this case physical examination will show decrease in apex pulse‚ by percussion the heart would be slightly enlarged and a slight friction rub by auscultation on lower left sternal border. And then we will do diagnostic tests including ECG‚ echocardiography‚ chest X-ray‚ and also some lab tests. ECG might show ST elevation‚ and also there might be PR depression. To differentiate from myocardial infarction there won’t be any pathologic q wave
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second- or third-degree atrioventricular block. Sinus bradycardia may be caused by excessive vagal tone‚ decreased sympathetic tone‚ or anatomical changes. It is common in athletes and is relatively benign. It may even be beneficial in acute myocardial infarction (especially inferior). Pathological bradycardia may be symptomatic of a brain tumor‚ digitalis toxicity‚ heart block‚ or vagotonus. Cardiac output
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arteries‚ causing a decreased blood supply to the myocardial layer of the heart‚ and prevents the arteries from dilating. As a result the tissue is deprived of oxygen necessary to thrive‚ which in turn can lead to myocardial ischemia‚ leading to several types of infarctions. Depending on the area of the ischemia‚ there can be several complications. For example‚ a ST segment elevation myocardial infarction (STEMI) occurs when there is a myocardial insufficiency caused by an occlusion that has completely
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Retrieved March 24‚ 2013 from http://www.cdc.gov/diabetes/pubs/pdf/ndfs_2011.pdf Centers for Disease Prevention and Prevention. (2011b). Chronic disease prevention and health promotion Kirk‚ J. K. (2010). Hyperglycemia Management Using Insulin in the Acute Care Setting: Therapies and Strategies for Care in the Non-Critically Ill Patient Webster‚ N.R. & Galley‚ H.F. (2009). Does strict Glucose control improve outcome. Wong‚ C. (2011). Natural treatments for type 2 diabetes. Retrieved March 24‚ 2013
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References: Handford‚ A.‚ Nowak‚ T. (2004). Congestive heart failure. Pathophysiology: Concepts and Applications for Health Care Professionals. (pp. 269-277). Boston‚ MA: The McGraw Hill Companies Inc. Jarvis‚ C. (2011). Clinical portrait of heart failure. Physical Examination & Health Assessment. (pp. 486). St. Louis‚ MO:
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mortality ‚re-infarction ‚repeat revascularization ‚bleeding and kidney dysfunction
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