Green-top Guideline No. 7 October 2010 Antenatal Corticosteroids to Reduce Neonatal Morbidity and Mortality Antenatal Corticosteroids to Reduce Neonatal Morbidity and Mortality This is the fourth edition of this guideline‚ which was previously published in April 1996‚ December 1999 and February 2004.The previous guideline was entitled Antenatal Corticosteroids to Prevent Respiratory Distress Syndrome. 1. Purpose and scope The aim of this guideline is to provide up-to-date information
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STEVENS-JOHNSON SYNDROME: PATHOPHYSIOLOGY‚ ETIOLOGY‚ DIAGNOSIS AND MANAGEMENT. Roberto Carmona Florida International University Abstract: Steven Johnson Syndrome is an inmune disease charactherized by a detachment of the epidermis from dermis. It could be fatal and the pathophysilogy involves a complex hypersensitivity reaction with the participation of T lymphocytes that induce keratinocyte’s apoptosis. The syndrome can be cause by drigs‚ infections and malignancies. The diagnosis
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This is a case study of a 76 year old female patient who is suffering from congestive cardiac failure. She has past medical history of hypertension‚ chronic renal failure‚ type 2 diabetes mellitus and hyperlipidaemia. She has been admitted in hospital several times recently and she was discharged 11 days ago from emergency department. Now she is suffering from dyspnoea and she feels like she can not catch her breath due to congestion of lungs. Congestion of lungs occurred due to congestive heart
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seems that A.O is suffering from left heart failure. Dyspnea the medical term for difficulty breathing is often an indicator of left heart failure. The clinical manifestations including dyspnea are caused by pulmonary vascular congestion and poor systemic oxygen perfusion (Huether & McCance‚ 2012). Other clinical manifestations physicians find with individuals with left heart failure include‚ coughing up mucus‚ fatigue‚ and a decreased urine output. Physicians will also perform a physical examination
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NU 327-801Pathophysiological Bases of Nursing University of South Alabama College of Nursing Fall 2010 Pyelonephrosis: More than just a UTI Introduction The purpose of this paper is to evaluate the pathophysiology of pyelonephrosis and to correlate that pathophysiology to the symptoms that accompany this disease process. This paper will also correlate the treatments and interventions which are appropriate for the nursing management and facilitation of wellness. Defining Pyelonephrosis
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INTRODUCTION ACUTE APPENDICITIS Appendicitis (or epityphlitis) is a condition characterized by inflammation of the appendix. All cases require removal of the inflamed appendix‚ either by laparotomy or laparoscopy. Untreated‚ mortality is high‚ mainly because of peritonitis and shock.[1] Reginald Fitz first described acute appendicitis in 1886‚[2] and it has been recognized as one of the most common causes of severe acute abdominal pain worldwide. Location of the appendix in the digestive system
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* Decreased muscle mass – decreased production Cr; Vit D * Diabetes Type I or II * Severe HTN or peripheral vascular disease * Preexisting CKD or proteinuria * CHF * Cirrhosis * NSAIDS‚ ACEi‚ vasodilators * Sepsis Pathophysiology: * Depressed RBF kidneys vulnerable to further insults - iatrogenic renal injury most common * Common iatrogenic combinations: * Preexisting renal disease‚ radio contrast agents‚ aminoglycosides‚ atheroembolism‚ or cardiovascular
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Botwinski‚ C. (2001). Systemic inflammatory response syndrome. Neonatal Network 20(5)‚ 21-.28. Braun‚ C.‚ & Anderson‚ C. (2006). Pathophysiology: Functional alterations in human health. Philadelphia: Lippincott Williams & Wilkins. Kumar‚ V.‚ Abbas‚ A.‚ Fausto‚ N.‚ Robbins‚ S.‚ & Cotran‚ R. (2005). Robbins and cotran: Pathologic basis of disease (7th ed.). United kingdom: Elsevier Saunders. McCance‚ K. L.‚ & Huether‚ S.E. (2009). Study guide for pathophysiology: The biological basis
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obesity‚ and hypertension (Lewis‚ et al‚ 2007‚ p. 821). Other risk factors may include the physical structure of the heart. For example‚ a patient may have endured damage to the heart due to a myocardial infarction‚ or a heart attack. Pathophysiology: The heart is a vital organ in the human body. It functions to pump deoxygenated blood to the lungs and oxygenated blood to the rest of the body. Oxygenated blood is crucial to the functioning of the organs and perfusion of tissues‚ which in
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Proteus mirabilis is a small gram-negative bacteria bacilli and a facultative anaerobe. A facultative anaerobe is when an organism can live in the absence or presence of oxygen. It is also a prokaryote‚ a single celled organism lacking a cell membrane bound nucleus‚ and can be divided into bacteria and archaea. Proteus mirabilis is characterized by their motility‚ its ability to distinguish maltose‚ and its inability to distinguish lactose. Proteus has the ability to stretch itself out and secrete
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